Reference

Alanine aminopeptidase excretion in urine after amidotrizoate administration in diabetics

Alanine aminopeptidase excretion in urine after amidotrizoate administration in diabetics. Stammberger, K.; Thulin, H.; Schanzenbach, H. (Kliniksanatorium Schielo, Schielo, E. Ger.). Dtsch. Gesundheitswes., 32(12), 542-3 (German) 1977. CODEN: DEGEA3. DOCUMENT TYPE: Journal CA Section: 1 (Pharmacodynamics) Injection of the renal contrast medium amidotrizoate (I) [117-96-4] into patients with diabetic nephropathy caused a greater increase in alanine aminopeptidase [54465-38-2] activity in the urine than in healthy subjects or diabetics without nephropathy.

Some organic acids attenuate the effects of furosemide on the endocochlear potential

Some organic acids attenuate the effects of furosemide on the endocochlear potential. Rybak, Leonard P.; Whitworth, Craig (Sch. Med., SIU, Springfield, IL 62708, USA). Hear. Res., 26(1), 89-93 (English) 1987. CODEN: HERED3. ISSN: 0378-5955. DOCUMENT TYPE: Journal CA Section: 1 (Pharmacology) A series of org. acid transport inhibitors reduced the endocochlear potential (EP) decline produced by furosemide [54-31-9] in the chinchilla. Probenecid [57-66-9], salicylate [69-72-7] and penicillin G [61-33-6] were much more effective than novobiocin [303-81-1], meclofenamate [644-62-2], or diatrizoate [117-96-4]. Inhibitors of org.There are some commonly used reagents with their cas registry numbers 114-33-0 and 644-62-2 in this article. base transport, choline [62-49-7], and N-Me nicotinamide [114-33-0] had no effect on the furosemide-induced drop of the EP. These findings suggest that at least part of furosemide ototoxicity may be mediated by org. acid transport. .