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Detail of "137975-06-5"

  • CAS Number:
  • 137975-06-5
  • Name:

  • Benzamide,N-[4-[[5-(dimethylamino)-2,3,4,5-tetrahydro-1H-1-benzazepin-1-yl]carbonyl]phenyl]-2-methyl-

  • Superlist Name:
  • Mozavaptan
  • Molecular Structure:
  • Formula:
  • C27H29N3O2
  • Molecular Weight:
  • 427.54
  • Synonyms:
  • 5-(Dimethylamino)-1-[4-(2-methylbenzamido)benzoyl]-2,3,4,5-tetrahydro-1H-benzazepine;OPC 31260;
  • Density:
  • 1.21 g/cm3
  • Boiling Point:
  • 543 °C at 760 mmHg
  • Flash Point:
  • 282.2 °C
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CAS No.137975-06-5 MozavaptanCompetitive Product

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CAS No.137975-06-5 Mozavaptan

Assay:≥ 98.5%  Appearance:white to whi...

Chemical name: 5-(Dimethylamino)-1-[4-(2-methylbenzamido)benzoyl]-2,3,4,5-tetrahydro-1H-benzazepine Structural formula: Molecular formula: C27H29N3O2 Molecular weight: 427.54 Loss on drying:≤ 0.5% ; Residue on ignition:≤ 0.2% ; Heavy metal:≤ 20ppm ;

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CAS No.137975-06-5 Mozavaptan

137975-06-5

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CAS No.137975-06-5 Mozavaptan

Assay:98.5%+

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CAS No.137975-06-5 Mozavaptan

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CAS No.137975-06-5 Mozavaptan

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CAS No.137975-06-5 Mozavaptan

Assay:>99.5%  Appearance:White to off...  Package:According to...

single impurity:<0.1%

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CAS No.137975-06-5 Mozavaptan

Assay:99%

24 hours Email contact is available, your email will be answered at the first time

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CAS No.137975-06-5 Mozavaptan

Name: Mozavaptan Synonyms: 5-(Dimethylamino)-1-[4-(2-methylbenzamido)benzoyl]-2,3,4,5-tetrahydro-1H-benzazepine Molecular Formula:C27H29N3O2 Molecular Weight:427.54 CAS Registry Number:137975-06-5

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CAS No.137975-06-5 Mozavaptan

Mozavaptan

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CAS No.137975-06-5 Mozavaptan

Mozavaptan

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CAS No.137975-06-5 Mozavaptan

Mozavaptan Chemical name: 5-(Dimethylamino)-1-[4-(2-methylbenzamido)benzoyl]-2,3,4,5-tetrahydro-1H-benzazepine Molecular formula: C27H29N3O2 Molecular weight: 427.54 CAS: 137975-06-5 Appearance:white to white-like crystal powder . If you have any requirements ,plz

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CAS No.137975-06-5 Mozavaptan

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CAS No.137975-06-5 Mozavaptan

97% Min

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CAS No.137975-06-5 Mozavaptan

98.0%min

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Reference

Inhibition of renal cystic disease development and progression by a vasopressin V2 receptor antagonist
Inhibition of renal cystic disease development and progression by a vasopressin V2 receptor antagonist. Gattone, Vincent H.; Wang, Xiaofang; Harris, Peter C.; Torres, Vicente E. (Anatomy and Cell Biology, Indiana University School of Medicine, Indianapolis, IN 46202, USA). Nature Medicine (New York, NY, United States), 9(10), 1323-1326 (English) 2003 Nature Publishing Group. CODEN: NAMEFI. ISSN: 1078-8956.In this article, certain chemicals are used. Some of their cas registry numbers are 60-92-4 and 137975-06-5 DOCUMENT TYPE: Journal CA Section: 1 (Pharmacology) The polycystic kidney diseases (PKDs) are a group of genetic disorders causing significant renal failure and death in children and adults. There are no effective treatments. Two childhood forms, autosomal recessive PKD (ARPKD) and nephronophthisis (NPH), are characterized by collecting-duct cysts. We used animal models orthologous to the human disorders to test whether a vasopressin V2 receptor (VPV2R) antagonist, OPC31260, would be effective against early or established disease. Adenosine-3',5'-cyclic monophosphate (cAMP) has a major role in cystogenesis, and the VPV2R is the major cAMP agonist in the collecting duct. OPC31260 administration lowered renal cAMP, inhibited disease development and either halted progression or caused regression of established disease. These results indicate that OPC31260 may be an effective treatment for these disorders and that clin. trials should be considered. .
SIADH and other hyponatremic disorders: Diagnosis and therapeutic problems
SIADH and other hyponatremic disorders: Diagnosis and therapeutic problems. Saito, Toshikazu (Department Medicine Japan, Jichi Medical School, Tochigi, Japan). Japanese Journal of Nephrology, 38(10), 429-434 (English) 1996 Nippon Jinzo Gakkai. CODEN: NJGKAU. ISSN: 0385-2385.Some commonly used reagents like 9001-01-8 and 137975-06-5 are used in this experiment. DOCUMENT TYPE: Journal; General Review CA Section: 14 (Mammalian Pathological Biochemistry) Section cross-reference(s): 2 A review, with 22 refs. Hyponatremia is more frequently obsd. in elderly patients assocd. with exaggerated response of ADH release and attenuated response of renin-aldosterone system. Differentiation of SIADH and hyponatremia other than SIADH is essential in anticipating the therapeutic effect of water restriction to correct hyponatremia. Serum concn. of uric acid and urinary excretion of kallikrein are significant parameters in biochem. discrimination index to differentiate these two types of hyponatremia. In addn. to sodium replacement, water restriction and mineral corticoid, newly developed nonpeptide ADH V2 receptor antagonist, OPC-31260, seems to be a useful tool to correct hyponatremia. .
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