CAS No.2034-22-2,1H-Imidazole,2,4,5-tribromo- Suppliers

Detail of "2034-22-2"

CAS Number:

2034-22-2

Name:

1H-Imidazole,2,4,5-tribromo-

Superlist Name:

2,4,5-Tribromoimidazole

Molecular Structure:

Formula:

C3H Br3 N2

Molecular Weight:

304.79

Synonyms:

Imidazole,2,4,5-tribromo- (7CI,8CI); 2,4,5-Tribromo-1H-imidazole;2,4,5-Tribromoimidazole; NSC 514965

EINECS:

217-997-5

Density:

2.782 g/cm³

Melting Point:

217-220 °C (dec.)(lit.)

Boiling Point:

401.3 °C at 760 mmHg

Flash Point:

196.5 °C

Appearance:

BEIGE-YELLOW POWDER

Hazard Symbols:

Risk Codes:

R23/24/25;R36/37/38

Safety:

Poison by ingestion and intravenous routes. When heated to decomposition it emits toxic fumes of Br⁻ and NO_x. Details

Transport Information:

UN 2811

Reference

Fire-retardant polyurethane composition: Fire-retardant polyurethane composition. Schulze, Heinz; Klein, Howard P. (Texaco Development Corp., USA). U.S. US 4032484 28 Jun 1977, 5 pp. (English). (United States of America). CODEN: USXXAM. CLASS: IC: C08G018-46. NCL: 260002500AJ. APPLICATION: US 76-650393 19 Jan 1976. DOCUMENT TYPE: Patent CA Section: 36 (Plastics Manufacture and Processing) 2,4,5-Tribromoimidazole (I) [2034-22-2] is an effective fireproofing agent for polyurethane foams. Thus, polyol (OH no. 454) 35.7, I 10.0, silicone 0.5, and isocyanate (PAPI) 40.8 parts were foamed to give articles with mech. properties comparable to foam prepd. without I. The foam contg. I had ASTM 1692 burn of 0.96 in./29.8 s while the foam without I had a burn of 3.3 in./60 s.

The toxicity and neuropathology of 2,4,5-tribromoimidazole and its derivatives in rats: The toxicity and neuropathology of 2,4,5-tribromoimidazole and its derivatives in rats. Verschoyle, R. D.; Brown, A. W.; Thompson, Carol A. (MRC Toxicol. Unit, Med. Res. Counc. Lab., Carshalton/Surrey SM5 4EF, UK). Arch. Toxicol., 56(2), 109-12 (English) 1984. CODEN: ARTODN. ISSN: 0340-5761. DOCUMENT TYPE: Journal CA Section: 4 (Toxicology) 2,4,5-Tribromoimidazole (I) [2034-22-2], I 1-n-butylcarboxylate [15287-51-1], and I 1-dimethylcarbamoyl [94847-66-2], when administered to rats, induced poisoning typical of uncouplers of oxidative phosphorylation. At 48 h, rats surviving a single toxic dose of 20-60 mg/kg developed permanent incoordination of the hindlimbs in the absence of brain edema. Neuropathol. examn. of the brain and spinal cord from perfused fixed rats at 24 h revealed neuronal necrosis and chromatolysis in the vestibular nucleus, the outer parietal neocortex, and the red nucleus. Chromatolysis and necrosis in these areas had increased at 72-96 h and were also obsd. in the deeper layers of the neocortex, the medial entorhinal cortex, the reticular formation, the grey matter of the spinal cord extending into the ventral horns, the dorsal and ventral cochlear nuclei, and the deep cerebellar nuclei, in decreasing order of severity. Neuronal necrosis was accompanied by an increased glial response, including neuronophagia, and at 16 days with astroglial hypertrophy and hyperplasia.

Detail of "2034-22-2"

1H-Imidazole,2,4,5-tribromo-

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CAS No.2034-22-2 2,4,5-Tribromoimidazole

CAS No.2034-22-2 2,4,5-Tribromoimidazole

CAS No.2034-22-2 2,4,5-Tribromoimidazole

Reference