CAS No.3211-76-5,Butanoic acid,2-amino-4-(methylseleno)-, (2S)- Suppliers

Detail of "3211-76-5"

CAS Number:

3211-76-5

Name:

Butanoic acid,2-amino-4-(methylseleno)-, (2S)-

Superlist Name:

L-(+)-Selenomethionine

Molecular Structure:

Formula:

C₅H₁₁NO₂Se

Molecular Weight:

196.11

Synonyms:

Butanoic acid,2-amino-4-(methylseleno)-, (S)-;Butyric acid, 2-amino-4-(methylselenyl)-, L-(8CI);L-Selenomethionine;Seleno-L-methionine;Selenomethionine;

Melting Point:

265 °C

Boiling Point:

320.78 °C at 760 mmHg

Flash Point:

147.803 °C

Solubility:

H2O: 50 mg/mL

Appearance:

white to off-white powder

Hazard Symbols:

Risk Codes:

23/25-33-50/53

Safety:

20/21-28-45-60-61-28A Details

Transport Information:

UN 3283 6.1/PG 2

Reference

An evaluation of protection offered by various dietary supplements against experimentally induced selenium-vitamin E deficiency in ducklings: An evaluation of protection offered by various dietary supplements against experimentally induced selenium-vitamin E deficiency in ducklings. Van Vleet, John F. (Sch. Vet. Med., Purdue Univ., West Lafayette, Indiana, USA). Am. J. Vet. Res., 38(8), 1231-6 (English) 1977. CODEN: AJVRAH. DOCUMENT TYPE: Journal CA Section: 18 (Animal Nutrition) Newly hatched male White Pekin ducklings fed a basal semisynthetic Se and vitamin E [1406-18-4] (Se-E)-deficient diet, developed, after 10 days, anorexia, slowed growth, reluctance to stand and move, and death. Gross lesions assocd. with the deficiency were pale or white chalky areas of myonecrosis and mineralization in the wall of the gizzard and intestine, pale streaks of degeneration in skeletal muscles, and pale or white chalky areas of necrosis and mineralization in the myocardium. Protection against the development of clin. signs or lesions of Se-E deficiency was provided, in groups fed the basal ration supplemented with 0.2 ppm of Se (as Na2SeO3 or selenomethionine [3211-76-5]), 100 IU .alpha.-tocopherol [59-02-9]/kg of diet, 0.3% ethoxyquin [91-53-2], or smaller amts. of these supplements if combined; in those fed com. duck starter mash; and in those fed low-Se pig feed (with or without Se supplementation). Partial protection resulted with smaller amts. of the aforementioned supplements. Ducklings suppplemented with methionine [63-68-3] or cystine [56-89-3] were not protected; the deficiency disease appeared to be enhanced in ducklings fed these diets. After 3 weeks, mean terminal hepatic Se content was maintained or increased in all Se-supplemented groups, including groups given only 0.05 ppm Se. Similar tissue accumulations of Se resulted from supplements of Na2SeO3 or selenomethionine. Tissue Se content was highest in liver and kidney, intermediate in heart and brain, and lowest in gizzard and skeletal muscle.

Detail of "3211-76-5"

Butanoic acid,2-amino-4-(methylseleno)-, (2S)-

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CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

CAS No.3211-76-5 L-(+)-Selenomethionine

Reference