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Detail of "4474-91-3"

  • CAS Number:
  • 4474-91-3
  • Name:
  • Angiotensin II,5-L-isoleucine-

  • Superlist Name:
  • Angiotensin II
  • Molecular Structure:
  • Formula:
  • C50H71N13O12
  • Molecular Weight:
  • 1046.34
  • Deleted CAS:
  • 3773-26-0,15963-93-6
  • Synonyms:
  • Angiotensin II, human (Octapeptide);Alanine,N-[1-[N-[N-[N-[N-(N2-L-a-aspartyl-L-arginyl)-L-valyl]-L-tyrosyl]-L-isoleucyl]-L-histidyl]-L-prolyl]-3-phenyl-,L- (6CI,7CI);
  • Density:
  • 1.43 g/cm3

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CAS No.4474-91-3 Angiotensin II

angiotensin II p-INNList-64,1990;r-INNList-31,1991; 5-L-isoleucineangiotensin II C50H71N13O12

Supplier:Tianjin Chicheng Chemicals Co.,ltd. [ China (Mainland)]

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CAS No.4474-91-3 Angiotensin II

Supplier:Jinan Haohua Industry CO., LTD [ China (Mainland)]

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CAS No.4474-91-3 Angiotensin II

Supplier:GenicBio Limited [ China (Mainland)]

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CAS No.4474-91-3 Angiotensin II

4474-91-3 H-Asp-Arg-Val-Tyr-Ile-His-Pro-Phe-OH 90937-05-6 H-Asp-Arg-Val-Tyr-Ile-p-amino-Phe-Pro-Phe-OH 53-73-6 H-Asn-Arg-Val-Tyr-Val-His-Pro-Phe-OH 52498-25-6 H-Arg-Val-Tyr-Ile-His-Pro-Ile-OH H-Asp-Arg-Val-3,5-diiodo-Tyr-Ile-His-Pro-Phe-OH 51833-69-3 Sar-Arg-Val-Tyr-Ile-His

Supplier:shanghai mocell biobech Co.,Ltd. [ China (Mainland)]

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CAS No.4474-91-3 Angiotensin II

Synonyms: DRVYIHPF;[ILE5]-ANGIOTENSIN 2;HYPERTENSION II HUMAN;L-ASP-ARG-VAL-TYR-ILE-HIS-PRO-PHE;H-ASP-ARG-VAL-TYR-ILE-HIS-PRO-PHE-OH;FLUORESCEIN-NHCS-NH-ASP-ARG-VAL-TYR-ILE-HIS-PRO-PHE-OH;ASP-ARG-VAL-TYR-ILE-HIS-PRO-PHE-OH 2CH3CHOOH H2O;ASP-ARG-VAL-TYR-ILE-HIS-PRO-PHE ACOH 4H2O

Supplier:Hybio Pharmaceutica Co.,Ltd [ China (Mainland)]

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Address:Hybio Medicine Park, No. 37, Keji C. Str 2nd Shenzhen Hi-tech Industrial Park 518057 P.R.China

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CAS No.4474-91-3 Angiotensin II

Creative Peptides is the world's leading provider for peptides and chemicals. We offer qualified products for 4474-91-3 (Angiotensin II Acetate). Please inquire us for 4474-91-3 (Angiotensin II Acetate).

Supplier:Creative Peptides [ United States]

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CAS No.4474-91-3 Angiotensin II

CBNumber: CB0338057 Chemical Name: ANGIOTENSIN II, HUMAN CAS No. 4474-91-3 Molecular Formula: C50H71N13O12 Formula Weight: 1046.18 Property storage temp. : ?20°C color : orange Safety WGK Germany : 3

Supplier:Cortex Biochem, Inc. [ United States]

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CAS No.4474-91-3 Angiotensin II

ANGIOTENSIN II, HUMAN

Supplier:NeoMPS SA [ France]

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CAS No.4474-91-3 Angiotensin II

ANGIOTENSIN II, HUMAN

Supplier:Nacalai Tesque, Inc. [ Japan]

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Address:Nijo Karasuma, Nakagyo-ku Kyoto 604-0855 Japan

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CAS No.4474-91-3 Angiotensin II

more information,pls contact with us!

Supplier:PEPTIDE-INST [ Japan]

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Address:4-1-2 Ina, MINOH-SHI, OSAKA 562-8686 JAPAN

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CAS No.4474-91-3 Angiotensin II

ANGIOTENSIN II, HUMAN

Supplier:SynPep Corporation [ Germany]

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CAS No.4474-91-3 Angiotensin II

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Supplier:POLYPEPTIDE [ Germany]

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Address:PolyPeptide Laboratories A/S 3400 Hiller?d Denmark

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CAS No.4474-91-3 Angiotensin II

ANGIOTENSIN II, HUMAN

Supplier:GenScript Corporation [ United States]

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CAS No.4474-91-3 Angiotensin II

more about it,please go to www.kanto.co.jp

Supplier:Kanto Chemical Co., Inc. [ Japan]

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CAS No.4474-91-3 Angiotensin II

Supplier:ChemPep, Inc. [ United States]

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Reference

Calcium reversal: inhibition by calcium ion of sustained contraction in calcium-free medium induced by various agonists in rat uterine smooth muscle
Calcium reversal: inhibition by calcium ion of sustained contraction in calcium-free medium induced by various agonists in rat uterine smooth muscle. Sakai, Kiyoshi; Higuchi-Nakamura, Kuniko; Uchida, Masaatsu Koujiro (Dep. Mol. Pharmacol., Meiji Coll. Pharm., Tokyo 154, Japan). Gen. Pharmacol., 16(2), 133-6 (English) 1985. CODEN: GEPHDP. ISSN: 0306-3623. DOCUMENT TYPE: Journal CA Section: 2 (Mammalian Hormones) Various agonists induced sustained contractions of estrogen-dominated rat uterine smooth muscle in Ca-free salt soln. contg. 0.2 mM EGTA after incubation of the muscle with 3 mM EGTA for 1 h. The magnitudes of contraction varied with agonists (bradykinin [58-82-2] > oxytocin [50-56-6] 3 arginine-vasopressin [113-79-1] > PGF2a [551-11-1] > 5-isoleucine-angiotensin II [4474-91-3] > acetylcholine [51-84-3] 3 PGE2 [363-24-6] 3 5-hydroxytryptamine [50-67-9]). Addn. of 10-4 M Ca2+ reduced the tension developed: Ca2+ inhibited these contractions when they were sufficiently large (marked inhibition on bradykinin-, oxytocin-, and vasopressin-induced contractions; definite one of PGF2a-induced contraction), as obsd. previously with oxytocin-induced contraction under the same conditions. This is termed Ca reversal. Thus, Ca reversal is not restricted to the oxytocin receptor-mediated response, but is apparently a general phenomenon.
Aldosterone responses to angiotensin II, adrenocorticotropin, and potassium in chronic experimental diabetes mellitus in rats
Aldosterone responses to angiotensin II, adrenocorticotropin, and potassium in chronic experimental diabetes mellitus in rats. Hayashi, Matsuhiko; Kitajima, Waichi; Saruta, Takao (Sch. Med., Keio Univ., Tokyo, Japan). Endocrinology (Baltimore), 115(6), 2205-9 (English) 1984. CODEN: ENDOAO. ISSN: 0013-7227. DOCUMENT TYPE: Journal CA Section: 2 (Mammalian Hormones) Section cross-reference(s): 14 The effects of isoleucine5-angiotensin II [4474-91-3], ACTH [9002-60-2], and K on aldosterone [52-39-1] secretion were examd. in conscious unrestrained streptozotocin-induced diabetic rats. In chronic exptl. diabetic rats, where plasma renin activity, plasma aldosterone concn., and urinary excretion of PGE2 were decreased, an attenuated response of aldosterone secretion was demonstrated after infusion of angiotensin II, ACTH, or K. Yet the plasma fluorogenic corticosteroids response to ACTH in diabetic rats was not different from that in control rats. After acute K infusion (0.30 mequiv/kg/min), plasma K levels in diabetic rats were higher than in control rats, although immunoreactive insulin levels remained unchanged compared to the elevation in control rats. Apparently, defects in aldosterone synthesis exist in chronic exptl. diabetic rats and K homeostasis is impaired during acute K loading. This change in K homeostasis may be related to both insulin and aldosterone deficiencies.
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