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Detail of "59-42-7"

  • CAS Number:
  • 59-42-7
  • Name:

  • Benzenemethanol,3-hydroxy-a-[(methylamino)methyl]-, (aR)-

  • Superlist Name:
  • Phenylephrine
  • Molecular Structure:
  • Formula:
  • C9H13NO2
  • Molecular Weight:
  • 167.23
  • Synonyms:
  • m-Methylaminoethanolphenol;m-Oxedrine;m-Sympathol;Benzenemethanol,3-hydroxy-a-[(methylamino)methyl]-, (R)-;Benzyl alcohol, m-hydroxy-a-[(methylamino)methyl]-, (-)- (7CI,8CI);(-)-Phenylephrine;(-)-m-Hydroxy-a-(methylaminomethyl)benzylalcohol;(-)-m-Oxedrine;(-)-m-Synephrine;(R)-(-)-Phenylephrine;(R)-Phenylephrine;L-Phenylephedrine;Mesaton;Mesatone;Metaoxedrin;Metaoxedrine;Metasympatol;Metasynephrine;Mezaton;Neo-Synephrine;Phenylephrine;R(-)-Mezaton;Visadron;l-m-Hydroxy-a-[(methylamino)methyl]benzylalcohol;m-Sympatol;m-Synephrine;
  • EINECS:
  • 200-424-8
  • Density:
  • 1.159 g/cm3
  • Melting Point:
  • 171°C
  • Boiling Point:
  • 341.1 °C at 760 mmHg
  • Flash Point:
  • 163.4 °C
  • Risk Codes:
  • 22-36/37/38
  • Safety:
  • 26-36-45 Details
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Phenylephrine Base We can supply the standard of EP6 .

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CAS No.59-42-7 Phenylephrine

product Name (-)-Phenylephrine Base ]Molecular Formula C9H13NO2 Molecular Weight 167.21 Specification:EP5 Packing: 25KG/drum Usage: adrenergic drugs to be used in surgery to extend the time for local anesthesia, nasal congestion, acute hypotension, septic embolism and a

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Reference

The relationship between stimulation-induced potassium release and amylase secretion in the mouse parotid
The relationship between stimulation-induced potassium release and amylase secretion in the mouse parotid. Petersen, O. H.; Gray, T. A.; Hall, R. A. (Dep. Physiol., Univ. Dundee, Dundee, Scot.). Pfluegers Arch., 369(3), 207-11 (English) 1977. CODEN: PFLABK. DOCUMENT TYPE: Journal CA Section: 2 (Hormone Pharmacology) The output of amylase [9000-92-4] from superfused mouse parotid segments in response to stimulation with acetylcholine (ACh) [51-84-3], phenylephrine (I) [59-42-7], and isoprenaline [7683-59-2] during exposure to solns. with varying K concns. was monitored by an on line automated fluorometric method. During stimulation with ACh or I a 10-fold increase in superfusion fluid K concn. caused an immediate very marked decrease in amylase output which was fully reversible. A 10-fold decrease in K concn. resulted in a prominent rise in amylase output. During stimulation with isoprenaline there was no effect on the amylase output of varying the extracellular K concn. ACh and I caused K release from the mouse parotid whereas isoprenaline had no such effect. Apparently, under conditions where stimulation-induced K release is enhanced there is also an enhanced amylase secretion and vice versa. There may therefore be a link between passive K transport and amylase secretion.
Pupil pharmacology in idiopathic headache
Pupil pharmacology in idiopathic headache. Fanciullacci, M.; Galli, P.; Monetti, M. G.; Pietrini, U.; Sicuteri, F. (Univ. Firenze, Florence, Italy). Boll. Soc. Ital. Biol. Sper., 52(23), 1950-1 (Italian) 1976. CODEN: BSIBAC. DOCUMENT TYPE: Journal CA Section: 1 (Pharmacodynamics) Section cross-reference(s): 14 The mydriatic response to fenfluramine (I) [458-24-2] (40 mg, orally) was lower in patients with severe idiopathic headache than in controls, whereas that to phenylephrine (II) [59-42-7] (conjunctival instillation) was greater. Considering the pharmacol. nature of the drugs, this different behavior indicated that the pupillary adrenergic nerve ending in patients with headache is deficient in noradrenaline [51-41-2].
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