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Detail of "84746-43-0"

  • CAS Number:
  • 84746-43-0
  • Name:
  • Small cardioactive peptide B

  • Molecular Structure:
  • Formula:
  • C52H80N14O11S2
  • Molecular Weight:
  • 1141.4088
  • Synonyms:
  • 18: PN: WO0114883SEQID: 30 claimed protein; Cardioactive peptide B (Aplysia californica); SCPB;Small cardioactive peptide B; Small cardioactive peptide B (Aplysiacalifornica); Small cardioexcitatory peptide B
  • Density:
  • 1.41 g/cm3
  • Appearance:
  • Solid

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CAS No.84746-43-0 Small cardioactive peptide B

Supplier:GenicBio Limited [ China (Mainland)]

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CAS No.84746-43-0 SCPB

SCPB

Supplier:NeoMPS SA [ France]

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Tel:+33 (0)3 88 79 08 79

Address:7 rue de Boulogne 67100 Strasbourg · France

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CAS No.84746-43-0 SCPB

SCPB

Supplier:GenScript Corporation [ United States]

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610

Tel:732 885 9188 025-84347317

Address:USA

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CAS No.84746-43-0 Small Cardioactive Peptide B (SCPB)

Supplier:Shanghai Apeptide Co., Ltd. [ China (Mainland)]

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Tel:+86-21-60871011 519-86330251

Address:Room405-406, JinhaiRoad, Pudong,Shanghai

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Reference

Endogenous peptides work at multiple sites in the nervous system in the control of gill behaviors in Aplysia
Endogenous peptides work at multiple sites in the nervous system in the control of gill behaviors in Aplysia. Lukowiak, Ken; Colmers, Wiliam F. (Fac. Med., Univ.Chemicals with cas numbers 113-80-4 and 84746-43-0 also play role. Calgary, Calgary, AB T2N 4N1, Can.). Regul. Pept., 17(2), 99-109 (English) 1987. CODEN: REPPDY. ISSN: 0167-0115. DOCUMENT TYPE: Journal CA Section: 12 (Nonmammalian Biochemistry) Section cross-reference(s): 2 The suprafusion of 2 endogenous neuropeptides, arginine vasotocin (AVT) and small cardioactive peptide B (SCPB) over the abdominal ganglion of A. californica affects the ability of a central gill motor neuron to elicit a gill withdrawal response. Gill motor neurons L7 or LDG1 were depolarized to produce the same no. of action potentials (APs) on each trial. When AVT (10-6 M) was suprafused, the motor neuron ability to elicit a gill movement was suppressed; whereas SCPB (10-6 M) superfusion facilitated the response. Neither peptide altered the passive membrane properties of the motor neurons nor did they affect the duration of their APs. Evidently, the peptides act via central control neurons which exert both suppressive and facilitatory control over gill reflex behaviors and assocd. neural activity. .
Colocalization of SCPB-like and FMRFamide-like immunoreactivities in crustacean nervous systems
Colocalization of SCPB-like and FMRFamide-like immunoreactivities in crustacean nervous systems. Callaway, Joseph C.; Masinovsky, Boris; Graubard, Katherine (Dep. Zool., Univ. Washington, Seattle, WA 98195, USA).There are some reagents with their cas registry numbers 84746-43-0 and 64190-70-1 are used in this study. Brain Res., 405(2), 295-304 (English) 1987. CODEN: BRREAP. ISSN: 0006-8993. DOCUMENT TYPE: Journal CA Section: 12 (Nonmammalian Biochemistry) A monoclonal antibody to the molluskan small cardioactive peptide SCPB and a polyclonal antibody to FMRF-amide were used to localize antigens in the stomatogastric nervous system and brain of 2 species of Cancer. Both antibodies labeled cell bodies, axons, and neuropilar processes in the brain and in the stomatogastric nervous system. All of the SCPB-immunoreactive neurons were colabeled with antibody to FMRF-amide. However, antibody to FMRF-amide labeled addnl. neurons of the commissural ganglion and the brain that were not immunoreactive to the monoclonal SCPB antibody. .
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