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119694-67-6

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119694-67-6 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 119694-67-6 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 1,1,9,6,9 and 4 respectively; the second part has 2 digits, 6 and 7 respectively.
Calculate Digit Verification of CAS Registry Number 119694-67:
(8*1)+(7*1)+(6*9)+(5*6)+(4*9)+(3*4)+(2*6)+(1*7)=166
166 % 10 = 6
So 119694-67-6 is a valid CAS Registry Number.

119694-67-6SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 19, 2017

Revision Date: Aug 19, 2017

1.Identification

1.1 GHS Product identifier

Product name [(E)-9H-pyrido[3,4-b]indol-3-ylmethylideneamino]thiourea

1.2 Other means of identification

Product number -
Other names 2-(9H-Pyrido(3,4-b)indol-3-ylmethylene)hydrazinecarbothioamide

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:119694-67-6 SDS

119694-67-6Downstream Products

119694-67-6Relevant articles and documents

Hybrid molecules: Growth inhibition of Leishmania donovani promastigotes by thiosemicarbazones of 3-carboxy-β-carbolines

Dodd,Ouannes,Robert-Gero,Potier

, p. 1272 - 1276 (1989)

The thiosemicarbazones of β-carboline-3-carboxaldehyde (compound 2) and 3-acetyl-β-carboline (compound 3) were found to effectively inhibit the in vitro growth of the promastigote form of Leishmania donovani, 50% inhibition being obtained at concentrations of 5.0 and 2.5 μM, respectively, while irreversible growth inhibition was achieved at 40 (compound 2) and 17.5 μM (compound 3). The thiosemicarbazone of pyridine-2-carboxaldehyde (compound 4) was considerably less active while both methyl β-carboline-3-carboxylate (compound 1) and the thiosemicarbazone of ethyl 5-formyl-6-azaindole-2-carboxylate (compound 5) were inactive at the highest concentrations tested. At concentrations provoking approximately 50% growth inhibition of promastigotes, compound 2 was observed to preferentially block DNA rather than RNA synthesis, but for compound 3, the reverse was true. Compound 3, the most active analogue studied, may thus act, at least partly, via a novel, though as yet unelucidated, mechanism.

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