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1-(allyloxy)-4-chloro-2-nitrobenzene is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

100246-31-9

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100246-31-9 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 100246-31-9 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 1,0,0,2,4 and 6 respectively; the second part has 2 digits, 3 and 1 respectively.
Calculate Digit Verification of CAS Registry Number 100246-31:
(8*1)+(7*0)+(6*0)+(5*2)+(4*4)+(3*6)+(2*3)+(1*1)=59
59 % 10 = 9
So 100246-31-9 is a valid CAS Registry Number.

100246-31-9Relevant academic research and scientific papers

Cyclizing radical carboiodination, carbotelluration, and carboaminoxylation of aryl amines

Hartmann, Marcel,Studer, Armido

supporting information, p. 8180 - 8183 (2014/08/18)

Radical carboiodination of various aryl amines is reported. Aryl diazonium salts, generated in situ from the corresponding aryl amines, are reacted with Bu4NI to provide the corresponding aryl radicals which undergo 5-exo or 6-exo cyclization. Iodine abstraction eventually affords the carboiodinated products in good to excellent yields. If TEMPO is added, the cascade provides the cyclized carboaminoxylation products. Running the reaction in the presence of PhTeTePh affords the phenyltellurated cyclized products.

Synthesis and in-vitro biological activity of macrocyclic urea Chk1 inhibitors

Li, Gaoquan,Tao, Zhi-Fu,Tong, Yunsong,Przytulinska, Magdalena K.,Kovar, Peter,Merta, Philip,Chen, Zehan,Zhang, Haiying,Sowin, Thomas,Rosenberg, Saul H.,Lin, Nan-Horng

, p. 6499 - 6504 (2008/03/18)

A variety of macrocyclic urea compounds were prepared as potent Chk1 inhibitors by modifying the C5 position of the benzene ring of the macrocyclic urea with ether moieties, aliphatic carbon chains, amide and halides. Enzymatic activity less than 20 nM was observed in 29 of 40 compounds. Compounds 14, 46d, and 48j provided the best overall results in the cellular assays as they abrogated doxorubicin-induced cell cycle arrest (IC50 = 3.31, 3.08, and 3.13 μM) and enhanced doxorubicin cytotoxicity (IC50 = 0.54, 1.27, and 0.96 μM) while displaying no single agent activity, respectively.

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