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1219940-63-2

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1219940-63-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1219940-63-2 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,2,1,9,9,4 and 0 respectively; the second part has 2 digits, 6 and 3 respectively.
Calculate Digit Verification of CAS Registry Number 1219940-63:
(9*1)+(8*2)+(7*1)+(6*9)+(5*9)+(4*4)+(3*0)+(2*6)+(1*3)=162
162 % 10 = 2
So 1219940-63-2 is a valid CAS Registry Number.

1219940-63-2Upstream product

1219940-63-2Downstream Products

1219940-63-2Relevant academic research and scientific papers

Synthesis of C-glycoside analogues of β-galactosamine-(1→4)-3-O-methyl-d-chiro-inositol and assay as activator of protein phosphatases PDHP and PP2Cα

Hans, Sunej K.,Camara, Fatoumata,Altiti, Ahmad,Martín-Montalvo, Alejandro,Brautigan, David L.,Heimark, Douglas,Larner, Joseph,Grindrod, Scott,Brown, Milton L.,Mootoo, David R.

, p. 1103 - 1110 (2010)

The glycan β-galactosamine-(1-4)-3-O-methyl-d-chiro-inositol, called INS-2, was previously isolated from liver as a putative second messenger-modulator for insulin. Synthetic INS-2 injected intravenously in rats is both insulin-mimetic and insulin-sensitizing. This bioactivity is attributed to allosteric activation of pyruvate dehydrogenase phosphatase (PDHP) and protein phosphatase 2Cα (PP2Cα). Towards identification of potentially metabolically stable analogues of INS-2 and illumination of the mechanism of enzymatic activation, C-INS-2, the exact C-glycoside of INS-2, and C-INS-2-OH the deaminated analog of C-INS-2, were synthesized and their activity against these two enzymes evaluated. C-INS-2 activates PDHP comparable to INS-2, but failed to activate PP2Cα. C-INS-2-OH was inactive against both phosphatases. These results and modeling of INS-2, C-INS-2 and C-INS-2-OH into the 3D structure of PDHP and PP2Cα, suggest that INS-2 binds to distinctive sites on the two different phosphatases to activate insulin signaling. Thus the carbon analog could selectively favor glucose disposal via oxidative pathways.

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