Cas 1308398-22-2,diethyl 2,6-dimethyl-4-(2-methyl-6-phenylimidazo[2,1-b]thiazol-5-yl)-1,4-dihydropyridine-3,5-dicarboxylate

1308398-22-2

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Basic information

Product Name: diethyl 2,6-dimethyl-4-(2-methyl-6-phenylimidazo[2,1-b]thiazol-5-yl)-1,4-dihydropyridine-3,5-dicarboxylate
Synonyms:
CAS NO:1308398-22-2
Molecular Formula:
Molecular Weight: 465.573
EINECS: N/A
Product Categories: N/A
Mol File: 1308398-22-2.mol
Article Data: 1

Chemical Properties

Melting Point: N/A
Boiling Point: N/A
Flash Point: N/A
Appearance: N/A
Density: N/A
Refractive Index: N/A
Storage Temp.: N/A
Solubility: N/A
CAS DataBase Reference: diethyl 2,6-dimethyl-4-(2-methyl-6-phenylimidazo[2,1-b]thiazol-5-yl)-1,4-dihydropyridine-3,5-dicarboxylate(CAS DataBase Reference)
NIST Chemistry Reference: diethyl 2,6-dimethyl-4-(2-methyl-6-phenylimidazo[2,1-b]thiazol-5-yl)-1,4-dihydropyridine-3,5-dicarboxylate(1308398-22-2)
EPA Substance Registry System: diethyl 2,6-dimethyl-4-(2-methyl-6-phenylimidazo[2,1-b]thiazol-5-yl)-1,4-dihydropyridine-3,5-dicarboxylate(1308398-22-2)

Safety Data

Hazard Codes: N/A
Statements: N/A
Safety Statements: N/A
WGK Germany:
RTECS:
HazardClass: N/A
PackingGroup: N/A
Hazardous Substances Data: 1308398-22-2(Hazardous Substances Data)

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Check Digit Verification of cas no

The CAS Registry Mumber 1308398-22-2 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,3,0,8,3,9 and 8 respectively; the second part has 2 digits, 2 and 2 respectively.
Calculate Digit Verification of CAS Registry Number 1308398-22:
(9*1)+(8*3)+(7*0)+(6*8)+(5*3)+(4*9)+(3*8)+(2*2)+(1*2)=162
162 % 10 = 2
So 1308398-22-2 is a valid CAS Registry Number.

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1308398-22-2Relevant articles and documents

Cystic fibrosis: A new target for 4-imidazo[2,1-b]thiazole-1, 4-dihydropyridines

Budriesi, Roberta,Ioan, Pierfranco,Leoni, Alberto,Pedemonte, Nicoletta,Locatelli, Alessandra,Micucci, Matteo,Chiarini, Alberto,Galietta, Luis J. V.

experimental part, p. 3885 - 3894 (2011/08/07)

The pharmacology of the cystic fibrosis transmembrane conductance regulator (CFTR) Cl- channel has attracted significant interest in recent years with the aim to search for rational new therapies for diseases caused by CFTR malfunction. Mutatio

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