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6-((3,5-dimethoxybenzyl)oxy)-2H-chromen-2-one is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

1318166-26-5

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1318166-26-5 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1318166-26-5 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,3,1,8,1,6 and 6 respectively; the second part has 2 digits, 2 and 6 respectively.
Calculate Digit Verification of CAS Registry Number 1318166-26:
(9*1)+(8*3)+(7*1)+(6*8)+(5*1)+(4*6)+(3*6)+(2*2)+(1*6)=145
145 % 10 = 5
So 1318166-26-5 is a valid CAS Registry Number.

1318166-26-5Relevant academic research and scientific papers

Inhibitory effects of 6-alkoxycoumarin and 7-alkoxycoumarin derivatives on lipopolysaccharide/interferon γ-stimulated nitric oxide production in RAW264 cells

Adfa, Morina,Itoh, Tomohiro,Hattori, Yosuke,Koketsu, Mamoru

experimental part, p. 963 - 966 (2012/09/21)

Coumarin and its derivatives are well known for their anti-inflammatory and anti-oxidative effects. In this study, we synthesized 32 coumarin derivatives from commercially available 6-hydroxycoumarin (6HC) and 7-hydroxycoumarin (7HC) and examined their ef

Structure-activity relationship and molecular mechanisms of ethyl 2-amino-6-(3,5-dimethoxyphenyl)-4-(2-ethoxy-2-oxoethyl)-4H -chromene-3- carboxylate (CXL017) and its analogues

Das, Sonia G.,Srinivasan, Balasubramanian,Hermanson, David L.,Bleeker, Nicholas P.,Doshi, Jignesh M.,Tang, Ruoping,Beck, William T.,Xing, Chengguo

scheme or table, p. 5937 - 5948 (2011/10/08)

Multidrug resistance (MDR) in cancer is a phenomenon in which administration of a single chemotherapeutic agent causes cross-resistance of cancer cells to a variety of therapies even with different mechanisms of action. Development of MDR against standard therapies is a major challenge in the treatment of cancer. Previously we have demonstrated a unique ability of CXL017 (5) to selectively target MDR cancer cells and synergize with mitoxantrone (MX) in HL60/MX2 MDR cells. Here we expand its scope and demonstrate that 5 can synergize with both vincristine and paclitaxel in three different MDR cell lines (HL60/DNR, K562/HHT300, and CCRF-CEM/VLB100). We also demonstrate that 5 has potent cytotoxicity in the NCI-60 panel of cell lines with an average IC 50 of 1.04 μM. In addition, 5 has a unique mechanism of action in comparison with standard agents in the NCI database based on COMPARE analysis. Further structure-activity relationship study led to the development of a more potent analogue, compound 7d, with an IC50 of 640 nM in HL60/MX2. Additionally, one enantiomer of 5 is 13-fold more active than the less active enantiomer. Taken together, our study has led to the discovery of a series of analogues that selectively target drug-resistant cancer cells with the potential for the treatment of drug-resistant cancers.

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