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N-methyl-N-<4-<1-<3-<(4-methylphenyl)sulfonyl>pyrrolidinyl>>-2-butynyl>acetamide is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

132377-31-2

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132377-31-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 132377-31-2 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 1,3,2,3,7 and 7 respectively; the second part has 2 digits, 3 and 1 respectively.
Calculate Digit Verification of CAS Registry Number 132377-31:
(8*1)+(7*3)+(6*2)+(5*3)+(4*7)+(3*7)+(2*3)+(1*1)=112
112 % 10 = 2
So 132377-31-2 is a valid CAS Registry Number.

132377-31-2Downstream Products

132377-31-2Relevant academic research and scientific papers

Muscarinic Receptor Binding and Activation of Second Messengers by Substituted N-Methyl-N-acetamides

Bradbury, Barton J.,Baumgold, Jesse,Paek, Robert,Kammula, Udai,Zimmet, Jeff,Jacobson, Kenneth A.

, p. 1073 - 1079 (2007/10/02)

A series of substituted azacycloalkyl analogues of the muscarinic agonist UH 5 (N-methyl-N-acetamide, 1a) were synthesized and evaluated pharmacologically.These compounds were developed as intermediates for further derivatization leading to functionalized congeners of 1a.The compounds were synthesized by using a Mannich-type condensation of N-acetyl-N-methylpropargylamine to various substituted saturated azaheterocycles.The compounds were screened at a single concentration in competitive binding assays in rat cerebral cortical membranes against either N-methylscopolamine (at 100 μM) or oxotremorine-M (at 1 μM) labels.Candidates were then selected for further evaluation of their effect on phosphoinositide (PI) turnover in membranes from A9L cells transfected with cDNA of either m1-muscarinic cholinergic receptors (m1AChRs) or m3AChRs.The analogues were also tested for the inhibition of adenylate cyclase in NG108-15 cells expressing m4AChRs.The azetidine analogue of 1a had a Ki value of 12 nM for the inhibition of oxotremorine-M binding in rat brain and had an agonist potency at m1-, m3-, and m4AChRs comparable to 1a.The substituted 5- and 6-member ring analogues generally had lower binding affinities and were less potent than 1a in stimulating PI turnover.Several compounds were moderately effective in inhibiting cyclic AMP production in NG108-15 cells.

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