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1354328-15-6

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1354328-15-6 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1354328-15-6 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,3,5,4,3,2 and 8 respectively; the second part has 2 digits, 1 and 5 respectively.
Calculate Digit Verification of CAS Registry Number 1354328-15:
(9*1)+(8*3)+(7*5)+(6*4)+(5*3)+(4*2)+(3*8)+(2*1)+(1*5)=146
146 % 10 = 6
So 1354328-15-6 is a valid CAS Registry Number.

1354328-15-6Upstream product

1354328-15-6Downstream Products

1354328-15-6Relevant articles and documents

Alpha-galactosylceramide analogs, a preparation method thereof, and a pharmaceutical composition for treatment and prevention of diseases by abnormal immune modulation comprising the same

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Paragraph 0104; 0137-0140; 0210; 0228-0231, (2019/07/10)

The present invention refers to alpha - ceramide analogs, including manufacturing method of immunomodulatory or pharmaceutical composition for treating or preventing diseases caused and relates to, more particularly NKT intracellular Th1 or selective activation reaction Th2 can be effective for treating or preventing diseases caused immunomodulatory or alpha - ceramide analogs, and manufacturing method of including a pharmaceutical composition for treating or preventing diseases caused immunomodulatory or more are disclosed. (by machine translation)

Heteroaromatic moieties in the sphingosine backbone of α- Galactosylceramides for noncovalent interactions with CD1d

Kim, Yongju,Kim, Jonghoon,Oh, Keunhee,Lee, Dong-Sup,Park, Seung Bum

supporting information; experimental part, p. 151 - 154 (2012/04/04)

A series of α-GalCer analogues containing heterocyclic and aromatic moieties in the sphingosine backbone were synthesized to improve the selectivity in the Th1/Th2 cytokine profile via noncovalent interaction with three aromatic residues at the binding pocket of CD1d. In vitro and in vivo biological evaluations revealed the treatment of α-GalCer analogue (6) induced the selective stimulation of natural killer T cells to facilitate the secretion of Th2 cytokines.

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