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1433974-79-8

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1433974-79-8 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1433974-79-8 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,4,3,3,9,7 and 4 respectively; the second part has 2 digits, 7 and 9 respectively.
Calculate Digit Verification of CAS Registry Number 1433974-79:
(9*1)+(8*4)+(7*3)+(6*3)+(5*9)+(4*7)+(3*4)+(2*7)+(1*9)=188
188 % 10 = 8
So 1433974-79-8 is a valid CAS Registry Number.

1433974-79-8Downstream Products

1433974-79-8Relevant academic research and scientific papers

CYCLIC N-ACYL O-AMINO PHENOL CBI DERIVATIVE

-

, (2014/10/15)

A group of cyclic N-acyl O-amino phenol CBI derivatives were synthesized and shown to be pro-drugs, subject to reductive activation by cleavage of a N-0 bond, effectively releasing the free drug in functional in vitro cellular assays for cytotoxic activity approaching the activity of the free drug, yet remain essentially stable to ex vivo DNA alkylation conditions. Assessment of the in vivo antitumor activity of a representative pro-drug indicates that a contemplated pro-drug approaches the potency and exceeds the efficacy of the free drug itself (CBI-indole2), indicating that the inactive pro-drugs not only effectively release the free drug in vivo, but that they offer additional advantages related to a controlled or targeted release in vivo.

Efficacious cyclic N-acyl O-amino phenol duocarmycin prodrugs

Wolfe, Amanda L.,Duncan, Katharine K.,Parelkar, Nikhil K.,Brown, Douglas,Vielhauer, George A.,Boger, Dale L.

, p. 4104 - 4115 (2013/07/05)

Two novel cyclic N-acyl O-amino phenol prodrugs are reported as new members of a unique class of reductively cleaved prodrugs of the duocarmycin family of natural products. These prodrugs were explored with the expectation that they may be cleaved selectively within hypoxic tumor environments that have intrinsically higher concentrations of reducing nucleophiles and were designed to liberate the free drug without the release of an extraneous group. In vivo evaluation of the prodrug 6 showed that it exhibits extraordinary efficacy (T/C > 1500, L1210; 6/10 one year survivors), substantially exceeding that of the free drug, that its therapeutic window of activity is much larger, permitting a dosing ≥40-fold higher than the free drug, and yet that it displays a potency in vivo that approaches the free drug (within 3-fold). Clearly, the prodrug 6 benefits from either its controlled slow release of the free drug or its preferential intracellular reductive cleavage.

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