144049-88-7

Basic information

• Product Name: kaempferol 3-O-(2''-O-β-D-glucopyranosyl)-α-L-rhamnopyranoside-nonaacetate
• CAS NO: 144049-88-7
• Molecular Weight: 972.862
• Mol File: 144049-88-7.mol
• Article Data: 1

Chemical Properties

• CAS DataBase Reference: kaempferol 3-O-(2''-O-β-D-glucopyranosyl)-α-L-rhamnopyranoside-nonaacetate(CAS DataBase Reference)
• NIST Chemistry Reference: kaempferol 3-O-(2''-O-β-D-glucopyranosyl)-α-L-rhamnopyranoside-nonaacetate(144049-88-7)
• EPA Substance Registry System: kaempferol 3-O-(2''-O-β-D-glucopyranosyl)-α-L-rhamnopyranoside-nonaacetate(144049-88-7)

Safety Data

• Hazardous Substances Data: 144049-88-7(Hazardous Substances Data)

Upstream product

• 108-24-7 acetic anhydride 
• 142451-65-8 3-O-[β-D-glucopyranosyl(1→2)]-α-L-rhamnopyranosyl kaempferol 

Relevant articles and documents

Selective responses of three Ginkgo biloba leaf-derived constituents on human intestinal bacteria.

The selective responses of Ginkgo biloba leaf-derived materials against six intestinal bacteria was examined using an impregnated paper disk method and compared with that of bilobalide, ginkgolides A and B, kaempferol, and quercetin. The components of G. biloba leaves were characterized as kaempferol 3-O-alpha-(6' -p-coumaroylglucosyl-beta-1,4-rhamnoside), kaempferol 3-O-(2' '-O-beta-D-glucopyranosyl)-alpha-L-rhamnopyranoside, and quercetin 3-O-alpha-(6' -p-coumaroylglucosyl-beta-1,4-rhamnoside) by spectroscopic analysis. The growth responses varied with each bacterial strain tested. At 2 mg/disk, kaempferol 3-O-alpha-(6' -p-coumaroylglucosyl-beta-1,4-rhamnoside) and quercetin 3-O-alpha-(6' -p-coumaroylglucosyl-beta-1,4-rhamnoside) revealed potent inhibition against Clostridium perfringens, and kaempferol 3-O-(2' '-O-beta-D-glucopyranosyl)-alpha-L-rhamnopyranoside showed a clear inhibitory effect on Escherichia coli. At 0.5 mg/disk, quercetin 3-O-alpha-(6' -p-coumaroylglucosyl-beta-1,4-rhamnoside) showed a strong activity against C. perfringens, but weak activity was exhibited by kaempferol 3-O-alpha-(6' -p-coumaroylglucosyl-beta-1,4-rhamnoside) against C. perfringens and kaempferol 3-O-(2' '-O-beta-D-glucopyranosyl)-alpha-L-rhamnopyranoside against E. coli. No inhibition was observed from treatments conducted with bilobalide, ginkgolides A and B, kaempferol, or quercetin. Furthermore, these isolated compounds did not inhibit Bifidobacterium bifidum, B. longum, B. adolescentis, or Lactobacillus acidophilus.