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1520856-93-2

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1520856-93-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1520856-93-2 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,5,2,0,8,5 and 6 respectively; the second part has 2 digits, 9 and 3 respectively.
Calculate Digit Verification of CAS Registry Number 1520856-93:
(9*1)+(8*5)+(7*2)+(6*0)+(5*8)+(4*5)+(3*6)+(2*9)+(1*3)=162
162 % 10 = 2
So 1520856-93-2 is a valid CAS Registry Number.

1520856-93-2Downstream Products

1520856-93-2Relevant academic research and scientific papers

Structure-based optimization of the terminal tripeptide in glycopeptide dendrimer inhibitors of pseudomonas aeruginosa biofilms targeting LecA

Kadam, Rameshwar U.,Bergmann, Myriam,Garg, Divita,Gabrieli, Gabriele,Stocker, Achim,Darbre, Tamis,Reymond, Jean-Louis

, p. 17054 - 17063 (2013)

The galactopeptide dendrimer GalAG2 ((β-Gal-OC6H 4CO-Lys-Pro-Leu)4(Lys-Phe-Lys-Ile)2Lys-His-Ile- NH2) binds strongly to the Pseudomonas aeruginosa (PA) lectin LecA, and it inhibits PA biofilms, as well as disperses already established ones. By starting with the crystal structure of the terminal tripeptide moiety GalA-KPL in complex with LecA, a computational mutagenesis study was carried out on the galactotripeptide to optimize the peptide-lectin interactions. 25 mutants were experimentally evaluated by a hemagglutination inhibition assay, 17 by isothermal titration calorimetry, and 3 by X-ray crystallography. Two of these tripeptides, GalA-KPY (dissociation constant (KD)=2.7 μM) and GalA-KRL (KD=2.7 μM), are among the most potent monovalent LecA ligands reported to date. Dendrimers based on these tripeptide ligands showed improved PA biofilm inhibition and dispersal compared to those of GalAG2, particularly G2KPY ((β-Gal-OC6H4CO-Lys-Pro-Tyr) 4(Lys-Phe-Lys-Ile)2Lys-His-Ile-NH2). The possibility to retain and even improve the biofilm inhibition in several analogues of GalAG2 suggests that it should be possible to fine-tune this dendrimer towards therapeutic use by adjusting the pharmacokinetic parameters in addition to the biofilm inhibition through amino acid substitutions. Breaking down biofilms: X-ray crystal-structure analysis of protein-ligand complexes and docking were used as the basis to optimize the terminal tripeptide of GalAG2, a tetravalent glycopeptide dendrimer inhibitor of the galactose-specific lectin LecA and of Pseudomonas aeruginosa biofilms. The study led to the discovery of new glycopeptide dendrimers with improved LecA binding, biofilm inhibition, and biofilm-dispersal properties (see figure for a close-up view of the GalA-WRI galactotripeptide bound to LecA).

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