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155814-23-6

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155814-23-6 Usage

Uses

3-Fluoro-2-methylcinnamic Acid is used in the preparation of antidepressants. 3-Fluoro-2-methylcinnamic Acid is also used in the preparation of arylvinylxanthines as selective A2 receptor antagonists.

Check Digit Verification of cas no

The CAS Registry Mumber 155814-23-6 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 1,5,5,8,1 and 4 respectively; the second part has 2 digits, 2 and 3 respectively.
Calculate Digit Verification of CAS Registry Number 155814-23:
(8*1)+(7*5)+(6*5)+(5*8)+(4*1)+(3*4)+(2*2)+(1*3)=136
136 % 10 = 6
So 155814-23-6 is a valid CAS Registry Number.

155814-23-6SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 13, 2017

Revision Date: Aug 13, 2017

1.Identification

1.1 GHS Product identifier

Product name 3-(3-fluoro-2-methylphenyl)prop-2-enoic acid

1.2 Other means of identification

Product number -
Other names 3-FLUORO-2-METHYLCINNAMIC ACID

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:155814-23-6 SDS

155814-23-6Relevant articles and documents

Design, synthesis and cytotoxic evaluation of novel imatinib amide derivatives that target Abl kinase

Yao, Ri-Sheng,Guan, Qiu-Xiang,Lu, Xiao-Qin,Ruan, Ban-Feng

, p. 20 - 28 (2015/03/31)

Novel imatinib amide derivatives (a1-28, b1-9) were synthesized and evaluated for their biological activities. All compounds were characterized by 1H NMR, MS and elemental analysis. Among all the derivatives, compounds a4, a10, a21, b1 and b2 displayed the most significant ability of inhibiting K562 cell proliferation with the IC50 values of 0.67, 0.66, 0.65, 0.59 and 0.62 μM, respectively, indicating that these compounds were potent inhibitors of Bcr-Abl in leukemic K562 cells, comparable to the reference compound imatinib. Molecular docking study was performed to position compounds a21 and b1 into the active site of Abl to determine the probable binding modes

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