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173216-42-7

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173216-42-7 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 173216-42-7 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 1,7,3,2,1 and 6 respectively; the second part has 2 digits, 4 and 2 respectively.
Calculate Digit Verification of CAS Registry Number 173216-42:
(8*1)+(7*7)+(6*3)+(5*2)+(4*1)+(3*6)+(2*4)+(1*2)=117
117 % 10 = 7
So 173216-42-7 is a valid CAS Registry Number.

173216-42-7Downstream Products

173216-42-7Relevant articles and documents

Sulfonium ion-promoted traceless Schmidt reaction of alkyl azides

Ardiansah, Bayu,Kakiuchi, Kiyomi,Morimoto, Tsumoru,Tanimoto, Hiroki,Tomohiro, Takenori

, p. 8738 - 8741 (2021/09/08)

Schmidt reaction by sulfonium ions is described. General primary, secondary, and tertiary alkyl azides were converted to the corresponding carbonyl or imine compounds without any trace of the activators. This bond scission reaction through 1,2-migration of C-H and C-C bonds was accessible to the one-pot substitution reaction.

Small Molecule Microarray Based Discovery of PARP14 Inhibitors

Peng, Bo,Thorsell, Ann-Gerd,Karlberg, Tobias,Schüler, Herwig,Yao, Shao Q.

supporting information, p. 248 - 253 (2016/12/30)

Poly(ADP-ribose) polymerases (PARPs) are key enzymes in a variety of cellular processes. Most small-molecule PARP inhibitors developed to date have been against PARP1, and suffer from poor selectivity. PARP14 has recently emerged as a potential therapeutic target, but its inhibitor development has trailed behind. Herein, we describe a small molecule microarray-based strategy for high-throughput synthesis, screening of >1000 potential bidentate inhibitors of PARPs, and the successful discovery of a potent PARP14 inhibitor H10 with >20-fold selectivity over PARP1. Co-crystallization of the PARP14/H10 complex indicated H10 bound to both the nicotinamide and the adenine subsites. Further structure–activity relationship studies identified important binding elements in the adenine subsite. In tumor cells, H10 was able to chemically knockdown endogenous PARP14 activities.

Nickel Mediated Reduction of Azides by Bu3SnH

Malanga, Corrado,Mannucci, Serena,Lardicci, Luciano

, p. 701 - 715 (2007/10/03)

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