24552-02-1

Basic information

• Product Name: N-Methyl-N-octylnonylamine
• Synonyms: N-Methyl-N-octyl-1-nonanamine;N-Methyl-N-octylnonylamine
• CAS NO: 24552-02-1
• Molecular Formula: C18H39N
• Molecular Weight: 269.51
• Mol File: 24552-02-1.mol

Chemical Properties

• Appearance: /
• CAS DataBase Reference: N-Methyl-N-octylnonylamine(CAS DataBase Reference)
• NIST Chemistry Reference: N-Methyl-N-octylnonylamine(24552-02-1)
• EPA Substance Registry System: N-Methyl-N-octylnonylamine(24552-02-1)

Safety Data

• Hazardous Substances Data: 24552-02-1(Hazardous Substances Data)

Upstream product

• 693-58-3 1-Bromononane 
• 111-83-1 1-bromo-octane 
• 39093-27-1 n-nonyl-n-methylamine 

Relevant articles and documents

Puerarin derivative with high biological activity as well as preparation method and application thereof

The invention relates to a puerarin derivative as well as a preparation method and application thereof. The puerarin derivative is characterized in that puerarin is used as a raw material; through five-step reaction, a compound 6 is prepared; then, the co

Synthesis, SAR and pharmacological characterization of novel anthraquinone cation compounds as potential anticancer agents

Emodin, a natural anthraquinone derivative isolated from Rheum palmatum L., has been demonstrated to exhibit good anti-cancer effect. In this study, a series of novel quaternary ammonium salts of emodin, anthraquinone and anthrone were synthesized and their anticancer activities were tested in vitro. The effects of emodin quaternary ammonium salts on cell viability, apoptosis, intracellular ROS, and mitochondrial membrane potential were investigated in A375, BGC-823, HepG2 and HELF cells. The results demonstrated that compound 4a induced morphological changes and decreased cell viability. Apoptosis triggered by compound 4a was visualized using DAPI staining and Annexin V-FITC/PI staining. Compound 4a-induced apoptosis of A375 cells were showed to be associated with the dissipation of mitochondrial membrane potential (ΔΨm) as a result of the up-regulation of P53 and Caspase-3. When cancer cells were treated with emodin derivative, their ability to generate reactive oxygen species (ROS) rose significantly and the mitochondrial membrane potential decreased. Additionally, confocal microscopy assay confirmed that compound 4a was primarily located in the mitochondria of A375 cells. These results suggested that compound 4a has the potential for use in cancer therapy.