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424798-17-4

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424798-17-4 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 424798-17-4 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 4,2,4,7,9 and 8 respectively; the second part has 2 digits, 1 and 7 respectively.
Calculate Digit Verification of CAS Registry Number 424798-17:
(8*4)+(7*2)+(6*4)+(5*7)+(4*9)+(3*8)+(2*1)+(1*7)=174
174 % 10 = 4
So 424798-17-4 is a valid CAS Registry Number.

424798-17-4Downstream Products

424798-17-4Relevant academic research and scientific papers

Discovery and biological evaluation of 5-aryl-2-furfuramides, potent and selective blockers of the Nav1.8 sodium channel with efficacy in models of neuropathic and inflammatory pain

Kort, Michael E.,Drizin, Irene,Gregg, Robert J.,Scanio, Marc J. C.,Shi, Lei,Gross, Michael F.,Atkinson, Robert N.,Johnson, Matthew S.,Pacofsky, Gregory J.,Thomas, James B.,Carroll, William A.,Krambis, Michael J.,Liu, Dong,Shieh, Char-Chang,Zhang, XuFeng,Hernandez, Gricelda,Mikusa, Joseph P.,Zhong, Chengmin,Joshi, Shailen,Honore, Prisca,Roeloffs, Rosemarie,Marsh, Kennan C.,Murray, Bernard P.,Liu, Jinrong,Werness, Stephen,Faltynek, Connie R.,Krafte, Douglas S.,Jarvis, Michael F.,Chapman, Mark L.,Marron, Brian E.

, p. 407 - 416 (2008/09/18)

Nav1.8 (also known as PN3) is a tetrodotoxin-resistant (TTx-r) voltage-gated sodium channel (VGSC) that is highly expressed on small diameter sensory neurons and has been implicated in the pathophysiology of inflammatory and neuropathic pain. Recent studies using an NavL8 antisense oligonucleotide in an animal model of chronic pain indicated that selective blockade of Nav1.8 was analgesic and could provide effective analgesia with a reduction in the adverse events associated with nonselective VGSC blocking therapeutic agents. Herein, we describe the preparation and characterization of a series of 5-substituted 2-furfuramides, which are potent, voltage-dependent blockers (IC50 v1.8 channel. Selected derivatives, such as 7 and 27, also blocked TTx-r sodium currents in rat dorsal root ganglia (DRG) neurons with comparable potency and displayed > 100-fold selectivity versus human sodium (Na v1.2, Nav1.5, Nav1.7) and human ether-a-go-go (hERG) channels. Following systemic administration, compounds 7 and 27 dose-dependently reduced neuropathic and inflammatory pain in experimental rodent models.

Discovery of potent furan piperazine sodium channel blockers for treatment of neuropathic pain

Drizin, Irene,Gregg, Robert J.,Scanio, Marc J.C.,Shi, Lei,Gross, Michael F.,Atkinson, Robert N.,Thomas, James B.,Johnson, Matthew S.,Carroll, William A.,Marron, Brian E.,Chapman, Mark L.,Liu, Dong,Krambis, Michael J.,Shieh, Char-Chang,Zhang, XuFeng,Hernandez, Gricelda,Gauvin, Donna M.,Mikusa, Joseph P.,Zhu, Chang Z.,Joshi, Shailen,Honore, Prisca,Marsh, Kennan C.,Roeloffs, Rosemarie,Werness, Stephen,Krafte, Douglas S.,Jarvis, Michael F.,Faltynek, Connie R.,Kort, Michael E.

, p. 6379 - 6386 (2008/12/22)

The synthesis and pharmacological characterization of a novel furan-based class of voltage-gated sodium channel blockers is reported. Compounds were evaluated for their ability to block the tetrodotoxin-resistant sodium channel Nav1.8 (PN3) as

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