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3-Ethyl-N,N,2-trimethyl-1H-indol-5-amine is a complex organic compound with the molecular formula C14H20N2. It is a derivative of indole, a heterocyclic aromatic organic compound that contains a benzene ring fused to a pyrrole ring. This specific compound features an ethyl group at the 3-position, two methyl groups at the 2-position, and a trimethyl group at the nitrogen atom. It is a colorless solid and is used as an intermediate in the synthesis of various pharmaceuticals and agrochemicals. Due to its complex structure and potential applications, it is an important compound in the field of organic chemistry.

576-11-4

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576-11-4 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 576-11-4 includes 6 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 3 digits, 5,7 and 6 respectively; the second part has 2 digits, 1 and 1 respectively.
Calculate Digit Verification of CAS Registry Number 576-11:
(5*5)+(4*7)+(3*6)+(2*1)+(1*1)=74
74 % 10 = 4
So 576-11-4 is a valid CAS Registry Number.
InChI:InChI=1/C13H18N2/c1-5-11-9(2)14-13-7-6-10(15(3)4)8-12(11)13/h6-8,14H,5H2,1-4H3

576-11-4Downstream Products

576-11-4Relevant academic research and scientific papers

2,3-Dialkyl(dimethylamino)indoles: Interaction with 5HT1, 5HT2, and rat stomach fundal serotonin receptors

Fludzinski,Wittenauer,Schenck,Cohen

, p. 2415 - 2418 (2007/10/02)

2,3-Dialkyl(dimethylamino)indoles, synthesized via the Fisher indole synthesis, were found to weakly bind to 5HT1 and 5HT2 sites in brain cortical membranes (IC50 greater than 1 μM at both sites for all compounds). These (dimethylamino)indoles were relatively potent antagonists of the serotonin receptor in the rat stomach fundus. At higher concentrations, several of the compounds were weak agonists at this receptor. For direct comparison with data obtained in the isolated rat fundus, antagonism of serotonin-induced contractions at 5HT2 receptors in the rat jugular vein was also examined. Several of the compounds showed good selectivity for the fundus receptor relative to the 5HT2 receptor; together with minimal affinity for 5HT1 and 5HT2 binding sites in brain cortical membranes, these results support the idea that the serotonin receptor in the stomach fundus is distinct from 5HT1 and 5HT2 binding sites.

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