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"Boc-Trp-Nle-Asp-Phe-NH2" is a peptide compound consisting of six amino acids: Boc-protected tryptophan (Trp), norleucine (Nle), aspartic acid (Asp), and phenylalanine (Phe), with an amide group (NH2) at the C-terminus. The Boc group is a protecting group for the amino group, which is commonly used in peptide synthesis to prevent unwanted side reactions. This specific sequence of amino acids may have potential applications in pharmaceuticals, as it could be a part of a larger peptide or protein with specific biological activity. The exact function or use of this peptide would depend on its context within a larger molecule or its interaction with biological targets.

6667-38-5

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6667-38-5 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 6667-38-5 includes 7 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 4 digits, 6,6,6 and 7 respectively; the second part has 2 digits, 3 and 8 respectively.
Calculate Digit Verification of CAS Registry Number 6667-38:
(6*6)+(5*6)+(4*6)+(3*7)+(2*3)+(1*8)=125
125 % 10 = 5
So 6667-38-5 is a valid CAS Registry Number.

6667-38-5Relevant academic research and scientific papers

CCK-B agonist or antagonist activities of structurally hindered and peptidase-resistant Boc-CCK4 derivatives

Corringer,Weng,Ducos,Durieux,Boudeau,Bohme,Roques

, p. 166 - 172 (2007/10/02)

Replacement of Met31 by (N-Me)Nle in CCK8 or CCK4 has been shown to improve the affinity and selectivity for CCK-B receptors. In order to obtain molecules with enhanced bioavailability, two novel series of protected tetrapeptides of the general formula Boc-Trp30-X-Asp-Y33 have been developed. Introduction of (N-Me)Nle and the bulky, aromatic naphthylalaninamide (Nal-NH2) in positions X and Y, respectively, does not greatly modify the affinity for guinea pig brain CCK-B receptors. In contrast, incorporation of hindering N-methyl amino acids such as (N-Me)Phe, (N-Me)Phg, or (N-Me)Chg, but not their non-methylated counterparts, in position X induced a large decrease in affinity for the CCK-B binding sites. Among the various peptides synthesized, Boc-[(N-Me)Nle31,1Nal- NH233]CCK4 (2) (K(I) = 2.8 nM), Boc-[Phg31,1Nal-NH233]CCK4 (15) (K(I) = 14 nM), and Boc-[Phg31,1Nal-N(CH3)233]CCK4 (17) (K(I) = 39 nM) displayed good affinities for brain CCK-B receptors and had good selectivity ratios. These pseudopeptides, in which the presence of unnatural and hydrophobic residues is expected to improve their penetration of the central nervous system, were shown to be very resistant to brain peptidases. Interestingly, whereas compounds 2 and 15 proved to be full agonists for rat hippocampal CCK-B receptors when measured in an electrophysiological assay, compound 17 behaved as a potent antagonist in the same test and displayed a good affinity in rat brain K(I)(CCK-B) = 51 nM as compared to the Merck antagonist L365,260, K(I)(CCK-B) = 12 nM. This illustrates a simple means to obtain CCK-B antagonists and suggests that the free, CONH2 group plays a critical role in the recognition of the agonist state of brain CCK-B receptors.

Synthesis and Biological Activity of CCK Heptapeptide Analogues. Effects of Conformational Constraints and Standard Modifications on Receptor Subtype Selectivity, Functional Activity in Vitro, and Appetite Suppression in Vivo

Holladay, Mark W.,Bennett, Michael J.,Tufano, Michael D.,Lin, C. W.,Asin, Karen E.,et al.

, p. 2919 - 2928 (2007/10/02)

A series of modifications of the CCK7 analogue (des-NH2)Tyr(SO3-)-Nle-Gly-Trp-Nle-Asp-Phe-NH2 was prepared and tested for binding to guinea pig CCK-A and CCK-B receptors and in CCK-A-mediated functional assays.Selected analogues also were teste

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