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1,2,2,2-Tetrachloro-N-benzyloxycarbonylethylamine is a complex organic compound with the molecular formula C10H12Cl4NO2. It is a derivative of ethylamine, featuring a benzyloxycarbonyl group attached to the nitrogen atom and four chlorine atoms substituted at the 1,2,2,2 positions. 1,2,2,2-tetrachloro-N-benzyloxycarbonylethylamine is primarily used as a protecting group in organic synthesis, particularly in peptide chemistry, to prevent unwanted side reactions and ensure the selective formation of desired products. The benzyloxycarbonyl (Z) group is a popular choice for protecting amino groups due to its stability and ease of removal under mild acidic conditions. The tetrachloro substituents in 1,2,2,2-tetrachloro-N-benzyloxycarbonylethylamine further enhance its stability and reactivity, making it a valuable tool in the synthesis of various pharmaceuticals and bioactive molecules.

6776-61-0

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6776-61-0 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 6776-61-0 includes 7 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 4 digits, 6,7,7 and 6 respectively; the second part has 2 digits, 6 and 1 respectively.
Calculate Digit Verification of CAS Registry Number 6776-61:
(6*6)+(5*7)+(4*7)+(3*6)+(2*6)+(1*1)=130
130 % 10 = 0
So 6776-61-0 is a valid CAS Registry Number.

6776-61-0Relevant academic research and scientific papers

Discovery of a dual tubulin polymerization and cell division cycle 20 homologue inhibitor via structural modification on apcin

Huang, Pan,Le, Xiangyang,Huang, Fei,Yang, Jie,Yang, Haofeng,Ma, Junlong,Hu, Gaoyun,Li, Qianbin,Chen, Zhuo

, p. 4685 - 4700 (2020/06/08)

Apcin is one of the few compounds that have been previously reported as a Cdc20 specific inhibitor, although Cdc20 is a very promising drug target. We reported here the design, synthesis, and biological evaluations of 2,2,2-trichloro-1-aryl carbamate derivatives as Cdc20 inhibitors. Among these derivatives, compound 9f was much more efficient than the positive compound apcin in inhibiting cancer cell growth, but it had approximately the same binding affinity with apcin in SPR assays. It is possible that another mechanism of action might exist. Further evidence demonstrated that compound 9f also inhibited tubulin polymerization, disorganized the microtubule network, and blocked the cell cycle at the M phase with changes in the expression of cyclins. Thus, it induced apoptosis through the activation of caspase-3 and PARP. In addition, compound 9f inhibited cell migration and invasion in a concentration-dependent manner. These results provide guidance for developing the current series as potential new anticancer therapeutics.

SEQUENTIAL CHIRALITY IN INTRAMOLECULAR AMIDOMERCURATION REACTIONS

Harding, Kenn E.,Hollingsworth, Donald R.,Reibenspies Joseph

, p. 4775 - 4778 (2007/10/02)

Control of absolute stereochemistry in the amination of allyl alcohol is effected by transfer chirality from a chiral auxiliray to the amidal carbon of an N-acylaminomethyl ether derivative, which upon intramolecular amidomercuration results in a second t

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