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3-(Acetylamino-methyl)-benzoic acid is a chemical compound with the molecular formula C10H11NO4. It is a derivative of benzoic acid, featuring an acetylamino-methyl group attached to the 3-position of the benzene ring. 3-(acetylamino-methyl)-benzoic acid is known for its potential applications in the synthesis of pharmaceuticals and other organic compounds. It is characterized by its ability to form salts and esters, which can be used in various chemical reactions. The compound's structure allows it to participate in a range of chemical transformations, making it a valuable intermediate in organic synthesis.

777-69-5

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777-69-5 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 777-69-5 includes 6 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 3 digits, 7,7 and 7 respectively; the second part has 2 digits, 6 and 9 respectively.
Calculate Digit Verification of CAS Registry Number 777-69:
(5*7)+(4*7)+(3*7)+(2*6)+(1*9)=105
105 % 10 = 5
So 777-69-5 is a valid CAS Registry Number.

777-69-5Relevant academic research and scientific papers

HETEROARYL COMPOUNDS AS IRAK INHIBITORS AND USES THEREOF

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Paragraph 00170, (2017/05/02)

The present invention relates to compounds of Formula (I) and pharmaceutically acceptable compositions thereof, useful as IRAK inhibitors.

Design and synthesis of benzoic acid derivatives as influenza neuraminidase inhibitors using structure-based drug design

Chand, Pooran,Babu, Yarlagadda S.,Bantia, Shanta,Chu, Naiming,Cole, L. Brent,Kotian, Pravin L.,Laver, W. Graeme,Montgomery, John A.,Pathak, Ved P.,Petty, Sandra L.,Shrout, David P.,Walsh, David A.,Walsh, Gerald M.

, p. 4030 - 4052 (2007/10/03)

A series of 94 benzoic acid derivatives was synthesized and tested for its ability to inhibit influenza neuraminidase. The enzyme-inhibitor complex structure was determined by X-ray crystallographic analysis for compounds which inhibited the enzyme. The most potent compound tested in vitro, 5 (4- (acetylamino)-3-guanidinobenzoic acid), had an IC50 = 2.5 x 10-6 M against N9 neuraminidase. Compound 5 was oriented in the active site of the neuraminidase in a manner that was not predicted from the reported active site binding of GANA (4) with neuraminidase. In a mouse model of influenza, 5 did not protect the mice from weight loss due to the influenza virus when dosed intranasally.

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