herbicide propaquizafop 95% 87%TC 10%EC CAS No.:111479-05-1 CAS NO.111479-05-1

specifications

propaquizafop herbicide 95% 86%/87%tc and 10%ec
best quality/price/service
professional package
fao standards

common name :propaquizafop (bsi, draft e-iso, (m) draft f-iso)
iupac name: 2-isopropylideneamino-oxyethyl (r)-2-[4-(6-chloroquinoxalin-2-yloxy)phenoxy]propionate
cas rn [111479-05-1]

physical chemistry
composition (r)- isomer. mol. wt. 443.9 m.f. c22h22cln3o5 form colourless crystals. m.p. 66.3 °c v.p. 4.4 ´ 10-7 mpa (25 ºc) (oecd 104); 1.3 ´ 10-7 mpa (20 °c) kow logp = 4.78 (25 ºc) henry 9.2 ´ 10-8 pa m3 mol-1 (20 °c, calc.) s.g./density 1.30 (20 ºc) solubility in water 0.63 g/m3 (20 ºc). in ethanol 59, acetone 730, toluene 630, n-hexane 37, n-octanol 16 (all in g/l, 25 ºc). stability stable ³2 y in closed container at room temperature. moderately stable at acid and neutral ph. rapidly hydrolysed under alkaline conditions. stable to u.v. light.

applications
biochemistry fatty acid synthesis inhibitor, by inhibition of acetyl coa carboxylase (accase). mode of action systemic post-emergence herbicide. absorbed by foliage and roots and translocated throughout the plant. treated grasses cease growth within 3-4 days, show chlorosis of younger plant tissues, followed by a progressive collapse of the entire plant 10-20 days later. uses control of a wide range of annual and perennial grasses (including sorghum halepense, agropyron repens and cynodon dactylon) in soya beans, cotton, sugar beet, potatoes, peanuts, peas, oilseed rape and vegetables, at 60-200 g/ha. phytotoxicity occasionally soya beans and peas may show some chlorotic and necrotic spots on treated leaves at higher dosage rates; this does not affect further vegetative growth or yield. not to be used on cucurbits. formulation types ec.

mammalian toxicology
oral acute oral ld50 for rats >5000, mice 3009 mg/kg. skin and eye acute percutaneous ld50 for rats >2000 mg/kg. non-irritant to skin; moderate eye irritation (rabbits). no allergic sensitisation in open epicutaneous test (guinea pigs); sensitising in maximisation test. inhalation lc50 (4 h) 2.5 mg/l air. noel (2 y) for rats and mice 1.5 mg/kg b.w. daily; (1 y) for dogs 20 mg/kg b.w. daily. adi 0.015 mg/kg b.w. toxicity class who (a.i.) iii (table 5) ec hazard (r43)

ecotoxicology
birds lc50 (10 d) for mallard ducks and (14 d) for bobwhite quail >6593 mg/kg feed. fish lc50 (96 h) for rainbow trout 1.2, carp 0.19, bluegill sunfish 0.34 mg/l. daphnia ec50 (acute) >>2 mg/l. algae ec50 (96 h) for selenastrum capricornutum >2.1 mg/l. bees ld50 (48 h) (oral) >20 mg/bee; (contact) >200 mg/bee. worms lc50 (14 d) >1000 mg/kg soil.

environmental fate
animals rapidly eliminated in the rat via urine and faeces, following oral administration. plants rapidly broken down in soya bean, sugar beet and cotton foliage to the free acid, followed by further metabolism to the quinoxaline oxyphenol. soil/environment fast degradation to the free acid (dt50 c. 3 d) and to further metabolites. dt50 (field) for parent and metabolites 15-26 d after spring application. dt50 for aquatic degradation and dissipation, for parent and metabolites, c. 20 d. no tendency for bioaccumulation