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Detail of "37339-90-5"

  • CAS Number:
  • 37339-90-5
  • Name:
  • Lentinan

  • Molecular Structure:
  • Formula:
  • (C42H70O35)n
  • Synonyms:
  • Biomoduline;LC 33;

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CAS No.37339-90-5 LentinanCompetitive Product

Assay:10:01  Appearance:fine powder  Package:92 kg/vacuum...Storage:store in a c...  Transportation:Air courier ...  Application:medicine add...

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CAS No.37339-90-5 Lentinan

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CAS No.37339-90-5 Lentinan

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CAS No.37339-90-5 Lentinan

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CAS No.37339-90-5 Lentinan

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CAS No.37339-90-5 Lentinan

Assay:LTN 10%,20%,...  Appearance:light brown ...  Package:25kg/drumStorage:2 years  Transportation:DHL/UPS/EMS/...  Application:Medicine, Fu...

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CAS No.37339-90-5 Lentinan

Assay:96.0-102.0%  Appearance:Brown powder  Package:5kg/bag or 2...

1.Name:Lentinan / 2.MF: C42H72O36 / 3.Grade Standard: Food Grade, Medicine Grade 4.Competitive price & Best quality & Prompt delivery

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CAS No.37339-90-5 Lentinan

What is Shiitake Mushroom Extract? Shiitake mushroom extract is effective in helping to suppress cancer recurrence, and in prolonging the lifespan of cancer patients. It also can be used as a nutritional supplement, in a supplement blend, or added to any number of herbal formu

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CAS No.37339-90-5 Lentinan

Name: Shitake Mushroom P.E. Latin name:Lentinus edodes Sing. Product category: Standard Herb Extracts Product Effectiveness: support healthy liver function Botanical Source: Lentinus edodes Sing. Used Part: Whole Herb Specifications: Polysaccharides 50% test by UV Appeara

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CAS No.37339-90-5 Lentinan

Chemical Name: LC 33;LENTINAN;biomoduline;LENTINAN (SHIITAKE MUSHROOM POLYSACCHARIDES);LENTINEX(R);lentinan vial CAS : 37339-90-5 Molecular Formula: (C42H70O35)n Molecular Weight : 257.72

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Model No.: SHT Product Name: Shiitake Mushroom P.E. Plant Origin: Lentinus edodes Specification: SHT-Polysaccharide 10%, 20%, 30% U

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If you interested in these product, pls seach us by google of 3w dot pharma-chemical dot com or contact me by email of selenite88(a)hotmail dot com Lentinan (Shiitake Mushroom Polysaccharides) CAS 37339-90-5 Formula:material safety data sheet Confirms to usp30 or EP 5 or C

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LENTINAN

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Reference

Antitumor activity of lentinan in murine syngeneic and autochthonous hosts and its suppressive effect on 3-methylcholanthrene-induced carcinogenesis
Antitumor activity of lentinan in murine syngeneic and autochthonous hosts and its suppressive effect on 3-methylcholanthrene-induced carcinogenesis. Suga, Tetsuya; Shiio, Tsuyoshi; Maeda, Yukiko Y.; Chihara, Goro (Natl. Cancer Cent. Res. Inst., Tokyo, Japan). Cancer Res., 44(11), 5132-7 (English) 1984. CODEN: CNREA8. ISSN: 0008-5472. DOCUMENT TYPE: Journal CA Section: 1 (Pharmacology) Section cross-reference(s): 4 The antitumor effect of lentinan [37339-90-5] in syngeneic and autochthonous tumor-host systems and its suppressive effect on 3-methylcholanthrene (MC) [56-49-5]-induced carcinogenesis were confirmed using DBA/2 and SWM/Ms hosts. The regressive activity of lentinan against the solid form of Sarcoma 180 was the most effective in DBA/2, SWM/Ms, or A/J mice and less effective in C3H/He or C57BL/6 mice. The growth of an syngeneic MC-induced DBA/2.MC.CS-1 fibrosarcoma (native and trypsinized) was markedly inhibited, and the regression of tumors was detected by the i.p. injection of min. amts. of lentinan into DBA/2 mice, which were the most suitable host in lentinan treatment. When DBA/2 mice were used, lentinan was also effective for even autochthonous primary tumors induced within 15 wk after MC inoculation, but less effective for tumors induced during the 16 to 36 wk after MC treatment. Lentinan showed a prominent suppressive effect in MC-induced carcinogenesis using DBA/2 and SWM/Ms mice but no effect when BALB/c, C57BL/6, or C3H/He mice were used. The timing of lentinan administration in the latter result was examd. using SWM/Ms mice, and lentinan, when it was given daily for 10 days after the third week of MC inoculation, was strikingly effective (33%), but not so effective (63%) when lentinan was given after the sixth week of MC treatment, compared with tumor-occurrence rate in the control group (88%). The reason why DBA/2, SWM/Ms, or A/J mice were suitable hosts for lentinan treatment is not clear, but the natural killer capability or phagocytic macrophage function in these strains seems to have no relation to lentinan action, because A/J mice are deficient in natural killer function, and in these strains of mice the phagocytic function of macrophages is weak. It may be quite possible that these strains of mice are most sensitive to delayed-type hypersensitivity and/or cytotoxic T-cell response in which T-cells and lentinan play important roles. The tumor-host systems presented here provide a good model in which lentinan retains an inhibitory capacity in syngeneic and autochthonous hosts, and such a model offers the possibility for further study of the host defense mechanism against cancer.
Immunotherpeutic agents for neoplasm and infection control
Immunotherpeutic agents for neoplasm and infection control. (Ajinomoto Co., Inc., Japan). Jpn. Kokai Tokkyo Koho JP 59095220 A2 1 Jun 1984 Showa, 7 pp. (Japanese). (Japan). CODEN: JKXXAF. CLASS: IC: A61K037-04. ICA: A61K035-74; A61K035-84; A61K037-00. APPLICATION: JP 82-205219 22 Nov 1982. DOCUMENT TYPE: Patent CA Section: 1 (Pharmacology) Section cross-reference(s): 15 Neoplasm and infection are controlled by human interleukin 1-formation potentiators, such as lentinan [37339-90-5], and by lymphokines such as interleukin 2, natural killer factor, macrophage-activating factor (MAF), T-cell-replacing factor, and interferon. For example, mice were s.c. injected with lymphoma and tumor L-5178 and i.v. injected with 25 mg lentinan on days, 7, 14, and 21. The tumors were isolated from the i.p. cavity on day 25 and incubated with MAF to show a marked decrease of tumor cells, in comparison to the control where the cells were incubated in the absence of MAF.
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