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Detail of "630-93-3"

  • MSDS Download
  • CAS Number:
  • 630-93-3
  • Name:

  • 2,4-Imidazolidinedione,5,5-diphenyl-, sodium salt (1:1)

  • Superlist Name:
  • Phenytoin sodium
  • Molecular Structure:
  • Formula:
  • C15H11N2NaO2
  • Molecular Weight:
  • 274.27
  • Deleted CAS:
  • 1421-15-4|143-75-9|8017-52-5
  • Synonyms:
  • 2,4-Imidazolidinedione,5,5-diphenyl-, monosodium salt (9CI);5,5-Diphenylhydantoin sodium;Aleviatin sodium;Difenin;Ditoin;Enkefal;Epanutin;Epilan D;Eptoin;Hydantin;M-toin;Phenyloin;Prompt;Sodium5,5-diphenyl-2,4-imidazolidinedione;Sodiumdiphenylhydantoin;Sodium phenytoin;
  • EINECS:
  • 211-148-2
  • Boiling Point:
  • 428.2 °C at 760 mmHg
  • Flash Point:
  • 212.8 °C
  • Solubility:
  • aqueous base: soluble
  • Hazard Symbols:
  • HarmfulXn
  • Risk Codes:
  • 22-43-62-63
  • Safety:
  • 22-36/37/39-45 Details
  • Transport Information:
  • UN 2811 6.1/PG 3
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CBNumber: CB2304645 Molecular Formula: C15H11N2NaO2 Formula Weight: 274.25 MOL File: Mol file

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Reference

Improvements in or relating to the detection of drugs
Improvements in or relating to the detection of drugs. De Faubert Maunder, Michael Joseph (United Kingdom Secretary of State For Industry, London, Engl.). Brit. GB 1485955 14 Sep 1977, 4 pp. (English). (United Kingdom). CODEN: BRXXAA. CLASS: IC: G01N021-06. APPLICATION: GB 74-903 8 Jan 1974. DOCUMENT TYPE: Patent CA Section: 4 (Toxicology) A hypnotic drug detection kit capable of simple use at a distance from a lab. was based on reacting the test sample with a single alk. soln. contg. Co(II) thiocyanate (I) [3017-60-5] and/or Cu(II) acetate [142-71-2] in polar solvents. E.g., samples contg. 20-50 mg of hypnotic, tested with 1 drop of a soln. contg. I (10 g/L) and 2,6-dimethylmorpholine (50 mL made up to 1 L with anhyd. MeOH), showed a pink to purple color with amylobarbitone [57-43-2], Na amylobarbitone [64-43-7], phenobarbitone [50-06-6], Ca cyclobarbitone [143-76-0], hexobarbitone [56-29-1], hydantoin [461-72-3], phenytoin Na [630-93-3], and glutethimide [77-21-4].
Isoniazid-induced inhibition in the biotransformation of [14C] diphenylhydantoin in rat
Isoniazid-induced inhibition in the biotransformation of [14C] diphenylhydantoin in rat. Buttar, H. S. (Health Prot. Branch, Health Welfare Canada, Ottawa, Ont., Can.). Res. Commun. Chem. Pathol. Pharmacol., 18(1), 35-57 (English) 1977. CODEN: RCOCB8. DOCUMENT TYPE: Journal CA Section: 1 (Pharmacodynamics) The effect of isoniazid (I) [54-85-3] (0, 10, 20 and 40 mg/kg, i.v.) on the biliary and urinary excretion of 14C-labeled Na diphenylhydantoin (II Na salt) [630-93-3] (50 mg/kg, i.v.) was investigated in biliary-fistulated rats during the 1st 5 h. I caused a dose-dependent redn. in the total biliary excretion of 14C without affecting its output in urine. After 5 h, maximal levels of radioactivity were recorded in the liver, followed by heart and brain. The glucuronides of II metabolites accounted for most of the 14C in 5 pooled bile; less than 1% of the injected dose was excreted as unmetabolized II. I administration caused the following significant dose-related changes in biliary excretion: increased excretion of the unconjugated principal I metabolite, 5-(p-hydroxyphenyl)-5-phenylhydantoin (HPPH) [2784-27-2]; decreased excretion of HPPH-glucuronide [53819-79-7]; and increased excretion of the glucuronide conjugates of the polar metabolites of II. The results suggest that the I-induced elevation of II-derived 14C in blood and its marked accumulation in brain, heart and liver is due to inhibition of p-hydroxylation of II and the glucuronidation of HPPH.
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