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1379779-22-2

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1379779-22-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1379779-22-2 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,3,7,9,7,7 and 9 respectively; the second part has 2 digits, 2 and 2 respectively.
Calculate Digit Verification of CAS Registry Number 1379779-22:
(9*1)+(8*3)+(7*7)+(6*9)+(5*7)+(4*7)+(3*9)+(2*2)+(1*2)=232
232 % 10 = 2
So 1379779-22-2 is a valid CAS Registry Number.

1379779-22-2Downstream Products

1379779-22-2Relevant academic research and scientific papers

CONDENSED TRICLYCLIC COMPOUNDS AS INHIBITORS OF HIV REPLICATION

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Page/Page column 39, (2013/07/05)

Compounds of formula (I) and pharmaceutical compositions thereof: wherein A1 A2 and A3 are each independently selected from the group consisting of N and CR3, wherein R1 is an optionally substituted heterocyclyl or an optionally substituted -(C1-6)alkyl-heterocyclyl, R2 is an optionally substituted aryl or an optionally subsisted heteroaryl, R4 is an optionally substituted aryl, an optionally substituted heterocyclyl or an optionally substituted heteroaryl, useful as an inbitor of HIV replication.

Discovery of XL413, a potent and selective CDC7 inhibitor

Koltun, Elena S.,Tsuhako, Amy Lew,Brown, David S.,Aay, Naing,Arcalas, Arlyn,Chan, Vicky,Du, Hongwang,Engst, Stefan,Ferguson, Kim,Franzini, Maurizio,Galan, Adam,Holst, Charles R.,Huang, Ping,Kane, Brian,Kim, Moon H.,Li, Jia,Markby, David,Mohan, Manisha,Noson, Kevin,Plonowski, Arthur,Richards, Steven J.,Robertson, Scott,Shaw, Kenneth,Stott, Gordon,Stout, Thomas J.,Young, Jenny,Yu, Peiwen,Zaharia, Cristiana A.,Zhang, Wentao,Zhou, Peiwen,Nuss, John M.,Xu, Wei,Kearney, Patrick C.

, p. 3727 - 3731 (2012/07/16)

CDC7 is a serine/threonine kinase that has been shown to be required for the initiation and maintenance of DNA replication. Up-regulation of CDC7 is detected in multiple tumor cell lines, with inhibition of CDC7 resulting in cell cycle arrest. In this paper, we disclose the discovery of a potent and selective CDC7 inhibitor, XL413 (14), which was advanced into Phase 1 clinical trials. Starting from advanced lead 3, described in a preceding communication, we optimized the CDC7 potency and selectivity to demonstrate in vitro CDC7 dependent cell cycle arrest and in vivo tumor growth inhibition in a Colo-205 xenograft model.

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