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  • 142426-07-1 Structure
  • Basic information

    1. Product Name: C15H13NOS
    2. Synonyms:
    3. CAS NO:142426-07-1
    4. Molecular Formula:
    5. Molecular Weight: 255.34
    6. EINECS: N/A
    7. Product Categories: N/A
    8. Mol File: 142426-07-1.mol
  • Chemical Properties

    1. Melting Point: N/A
    2. Boiling Point: N/A
    3. Flash Point: N/A
    4. Appearance: N/A
    5. Density: N/A
    6. Refractive Index: N/A
    7. Storage Temp.: N/A
    8. Solubility: N/A
    9. CAS DataBase Reference: C15H13NOS(CAS DataBase Reference)
    10. NIST Chemistry Reference: C15H13NOS(142426-07-1)
    11. EPA Substance Registry System: C15H13NOS(142426-07-1)
  • Safety Data

    1. Hazard Codes: N/A
    2. Statements: N/A
    3. Safety Statements: N/A
    4. WGK Germany:
    5. RTECS:
    6. HazardClass: N/A
    7. PackingGroup: N/A
    8. Hazardous Substances Data: 142426-07-1(Hazardous Substances Data)

142426-07-1 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 142426-07-1 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 1,4,2,4,2 and 6 respectively; the second part has 2 digits, 0 and 7 respectively.
Calculate Digit Verification of CAS Registry Number 142426-07:
(8*1)+(7*4)+(6*2)+(5*4)+(4*2)+(3*6)+(2*0)+(1*7)=101
101 % 10 = 1
So 142426-07-1 is a valid CAS Registry Number.

142426-07-1Downstream Products

142426-07-1Relevant articles and documents

Benzisothiazolones arrest the cell cycle at the G2/M phase and induce apoptosis in HeLa cells

Gopinath, Pushparathinam,Ramalingam, Krishnan,Muraleedharan, Kannoth Manheri,Karunagaran, Devarajan

, p. 749 - 752 (2013)

Anticancer activities of a series of benzisothiazolones having alkyl, aryl and aralkyl substituents on the nitrogen atom and the mechanistic basis of cytotoxicity are presented. Cellular responses like DNA laddering, disruption of mitochondrial membrane potential and caspase-3 activation on incubation of HeLa cells with representative compounds from this group suggested the induction of apoptosis through an intrinsic pathway. Their ability to arrest the cell cycle at the G2/M phase was confirmed by flow cytometric analysis.

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