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6-methoxy-3-(4-methoxyphenyl)-2-methylquinoline is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

1609013-08-2

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1609013-08-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1609013-08-2 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,6,0,9,0,1 and 3 respectively; the second part has 2 digits, 0 and 8 respectively.
Calculate Digit Verification of CAS Registry Number 1609013-08:
(9*1)+(8*6)+(7*0)+(6*9)+(5*0)+(4*1)+(3*3)+(2*0)+(1*8)=132
132 % 10 = 2
So 1609013-08-2 is a valid CAS Registry Number.

1609013-08-2Relevant academic research and scientific papers

Discovery of 3-phenylquinolinylchalcone derivatives as potent and selective anticancer agents against breast cancers

Tseng, Chih-Hua,Tzeng, Cherng-Chyi,Hsu, Chih-Yao,Cheng, Chih-Mei,Yang, Chia-Ning,Chen, Yeh-Long

, p. 306 - 319 (2015/06/02)

A number of 3-phenylquinolinylchalcone derivatives were synthesized and evaluated in vitro for their antiproliferative activities against three breast cancer cell lines (MCF-7, MDA-MB-231, and SKBR-3), and a non-cancer normal epithelial cell line (H184B5F

Discovery of 2-[2-(5-nitrofuran-2-yl)vinyl]quinoline derivatives as a novel type of antimetastatic agents

Tseng, Chih-Hua,Tzeng, Cherng-Chyi,Chiu, Chien-Chih,Hsu, Chih-Yao,Chou, Chon-Kit,Chen, Yeh-Long

, p. 141 - 148 (2015/02/19)

A number of 2-furanylvinylquinoline derivatives were synthesized and evaluated for antiproliferative activities against the growth of four cancer cell lines including non-small cell lung cancer (A549 and H1299), breast cancer (MCF-7 and MDA-MB-231) and normal diploid embryonic lung cell line (MRC-5). Among them, (E)-6-methoxy-3-(4-methoxyphenyl)-2-[2-(5-nitrofuran-2-yl)vinyl]quinoline (10c) was found low cytotoxic in all cancer cells and normal cell. The aim of this study was to investigate the anti-invasive and anti-metastatic activity of compound 10c in H1299 human lung cancer cells. In this study, compound 10c inhibited the migration and invasion of cells in a concentration-dependent manner by wound healing assay and transwell invasion assay. Furthermore, the inhibition of both phosphorylation of Akt and ERK by compound 10c may be critical for cell migration and this may result in the down-regulation of several factors associated with cellular migration, including β-catenin transcription factor, Bcl-2 and COX-2. Interestingly, the treatment of compound 10c did not affect the expression level but reduced the activities of the MMP-2 and -9. The phosphorylation level of stress-activated kinase p38 was significantly increased following compound 10c treatment. To sum up, compound 10c had potential to suppress the migration and invasion of H1299 cancer cells in vitro and it could serve as a promising drug for the treatment of cancer metastasis.

Synthesis, antiproliferative and anti-dengue virus evaluations of 2-aroyl-3-arylquinoline derivatives

Tseng, Chih-Hua,Lin, Chun-Kuang,Chen, Yeh-Long,Hsu, Chih-Yao,Wu, Huey-Nan,Tseng, Chin-Kai,Lee, Jin-Ching

, p. 66 - 76 (2014/05/06)

A number of 2-aroyl-3-arylquinoline derivatives was synthesized and evaluated for their anti-Dengue virus activity. Both 2-(hydroxyphenylmethyl)-3- (4-methoxyphenyl)quinoline (13a) and 2-(4-hydroxybenzoyl)-3-(4-hydroxyphenyl) quinoline (17) were found to significantly inhibit the DENV2 RNA expression in Huh-7-DV-Fluc cells with a potency approximately equal to that of ribavirin and the inhibition is in a dose-dependent manner. Compounds 13a and 17 reduced DENV replication in both viral protein and mRNA levels, and no significant cell cytotoxicity was detected, with greater than 50% viability of Huh-7-DV-Fluc cells at a concentration of 100 μM. However, significant cytotoxicity was detected for the positive ribavirin. In addition, we performed infectious assay to further verify the inhibitory activity of 13a and 17 on DENV replication in protein and RNA levels. On the other hand, compounds 19a-19c exhibited IC 50 values ranged from 4.47 to 8.68 μM against A549, H1299, MCF-7, and Huh-7 which were approximately equal potent to the positive topotecan. Structural optimization of lead compounds, 13a and 17, and their detailed molecular mechanism of action are ongoing.

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