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7-[2-(4-Fluoro-phenyl)-2-oxo-ethoxy]-2-phenyl-chromen-4-one is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

202138-76-9

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202138-76-9 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 202138-76-9 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 2,0,2,1,3 and 8 respectively; the second part has 2 digits, 7 and 6 respectively.
Calculate Digit Verification of CAS Registry Number 202138-76:
(8*2)+(7*0)+(6*2)+(5*1)+(4*3)+(3*8)+(2*7)+(1*6)=89
89 % 10 = 9
So 202138-76-9 is a valid CAS Registry Number.

202138-76-9Relevant academic research and scientific papers

162. Synthesis and evaluation of α-methylidene-γ-butyrolactone bearing flavone and xanthone moieties

Tzeng,Zhao,Chen

, p. 2337 - 2344 (1997)

In a search for inhibitors of platelet aggregation, a number of α- methylidene-γ-butyrolactone 5 and 6 bearing flavone or xanthone moieties, respectively, were synthesized and evaluated for their antiplatelet activity against thrombin(Thr)-, arachidonic-acid(AA)-, collagen(Col)-, and platelet- activating-factor(PAF)-induced aggregation in washed rabbit platelets. These compounds were synthesized from 7-hydroxyflavone (1) or 3-hydroxyxanthone (2) via O-alkylation (→ 3 and 4, resp.) and Reformatsky-type condensation (Scheme). Most of the flavone-containing α-methylidene-γ-butyrolactones 5a- d showed potent antiplatelet effects on AA- and Col-induced aggregation, while xanthone derivatives 6c-e were found to have the same pharmacological profile than aspirin in which only AA-induced aggregation was inhibited (Table 1). However, 6c-e were approximately three to ten times more potent that aspirin (Table 2). For the vasorelaxing effects, 5a was the only compound which exhibited significant inhibitory activity on the high-K+- medium, Ca2+-induced vasoconstriction (Table 3). Both 5a and 6a, with an aliphatic Me substituent at C(γ) of the lactone, were active against norepinephrine-induced phasic and tonic constrictions while their γ-aryl- substituted counterparts 5b-f and 6b-f were inactive.

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