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Benzaldehyde-(3-fluoro-phenylhydrazone) is a chemical compound derived from the condensation of benzaldehyde with 3-fluorophenylhydrazine. It is an organic molecule with a molecular formula of C14H12FN2O, featuring a benzaldehyde group and a 3-fluorophenylhydrazone moiety. benzaldehyde-(3-fluoro-phenylhydrazone) is characterized by its ability to form a hydrazone linkage, which is a common structural feature in many organic compounds. It is often used in the synthesis of various pharmaceuticals and organic compounds due to its unique reactivity and stability. The presence of the fluorine atom in the 3-fluorophenyl group imparts specific electronic and steric properties to the molecule, which can influence its chemical behavior and reactivity in various reactions.

351-26-8

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351-26-8 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 351-26-8 includes 6 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 3 digits, 3,5 and 1 respectively; the second part has 2 digits, 2 and 6 respectively.
Calculate Digit Verification of CAS Registry Number 351-26:
(5*3)+(4*5)+(3*1)+(2*2)+(1*6)=48
48 % 10 = 8
So 351-26-8 is a valid CAS Registry Number.

351-26-8Downstream Products

351-26-8Relevant academic research and scientific papers

Broad-Spectrum Antifungal Agents: Fluorinated Aryl- and Heteroaryl-Substituted Hydrazones

Thamban Chandrika, Nishad,Dennis, Emily K.,Brubaker, Katelyn R.,Kwiatkowski, Stefan,Watt, David S.,Garneau-Tsodikova, Sylvie

, p. 124 - 133 (2021)

Fluorinated aryl- and heteroaryl-substituted monohydrazones displayed excellent broad-spectrum activity against various fungal strains, including a panel of clinically relevant Candida auris strains relative to a control antifungal agent, voriconazole (VRC). These monohydrazones displayed less hemolysis of murine red blood cells than that of VRC at the same concentrations, possessed fungicidal activity in a time-kill study, and exhibited no mammalian cell cytotoxicity. In addition, these monohydrazones prevented the formation of biofilms that otherwise block antibiotic effectiveness and did not trigger the development of resistance when exposed to C. auris AR Bank # 0390 over 15 passages.

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