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3-hydroxy-5,9-dimethyl-dec-8-en-2-one is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

367522-58-5

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367522-58-5 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 367522-58-5 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 3,6,7,5,2 and 2 respectively; the second part has 2 digits, 5 and 8 respectively.
Calculate Digit Verification of CAS Registry Number 367522-58:
(8*3)+(7*6)+(6*7)+(5*5)+(4*2)+(3*2)+(2*5)+(1*8)=165
165 % 10 = 5
So 367522-58-5 is a valid CAS Registry Number.

367522-58-5Downstream Products

367522-58-5Relevant academic research and scientific papers

Synthesis of α-hydroxy ketones from terpene aldehydes

Schueler, Martin,Zorn, Holger,Slawin, Alexandra M. Z.,Berger, Ralf G.

, p. 2591 - 2600 (2004)

Unsymmetrically substituted α-hydroxy ketones possessing isoprenoid units within the molecule were synthesised from commercially available terpene aldehydes. The synthesis was applicable to α,β-unsaturated aldehydes and afforded the respective α-hydroxy-m

Inactivation of protein farnesyltransferase by active-site-targeted dicarbonyl compounds

Okolotowicz, Karl J.,Lee, Wei-Jen,Hartman, Rosemarie F.,Kim, Ann Y.,Ottersberg, Steven R.,Robinson Jr., Dale E.,Lefler, Scott R.,Rose, Seth D.

, p. 194 - 202 (2007/10/03)

Upon farnesylation by protein farnesyltransferase (FTase), key proteins become compartmentalized in cells. For example, cell membrane localization is essential for the mitogenic role of mutant Ras protein, which acts as a switch for cancer cell proliferation. We report that α-dicarbonyl compounds derived from the isoprenoid skeleton or other hydrophobic groups potently obstruct farnesylation of a Ras model peptide by human recombinant FTase in vitro. A geranyl-derived isoprenoid diketone, 5,9-dimethyl-8-decene-2,3-dione, at 17 μM caused a 62% reduction in FTase activity after 30 minutes. A farnesyl-derived isoprenoid diketone, 5,9,13-trimethyl-8,12-tetradecadiene-2,3-dione, at 93 μM caused a 94% reduction after 30 minutes. Other dicarbonyl compounds found to be effective against FTase in vitro were (±)-6-(camphorquinone-10-sulfonamido)-hexanoic acid, 4,4′-biphenyldiglyoxaldehyde, dehydroascorbic acid 6-palmitate, 2-oxododecanal, and phenylglyoxal. Higher concentrations of the α-dicarbonyl compound resulted in more rapid and more extensive inactivation. These findings demonstrate that α-dicarbonyl compounds targeted to FTase interfere with protein farnesylation in vitro and may lead to derivatives that have utility as chemotherapeutic agents.

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