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(E,E)-1,4-bis[3'-hydroxy-4'-(methanesulfonyloxy)styryl]benzene is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

491853-79-3

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491853-79-3 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 491853-79-3 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 4,9,1,8,5 and 3 respectively; the second part has 2 digits, 7 and 9 respectively.
Calculate Digit Verification of CAS Registry Number 491853-79:
(8*4)+(7*9)+(6*1)+(5*8)+(4*5)+(3*3)+(2*7)+(1*9)=193
193 % 10 = 3
So 491853-79-3 is a valid CAS Registry Number.

491853-79-3Downstream Products

491853-79-3Relevant academic research and scientific papers

Synthesis and 11c-labelling of (E,E)-1-(3′,4′-dihydroxystyryl)-4-(3′-methoxy-4 ′-hydroxystyryl) benzene for PET imaging of amyloid deposits

Wang, Yanming,Mathis, Chester A.,Huang, Guo-Feng,Holt, Daniel P.,Debnath, Manik L.,Klunk, William E.

, p. 647 - 664 (2007/10/03)

Carboxylic acid derivatives of the amyloid-binding dye Congo red do not enter the brain well and are thus unable to serve as in vivo amyloid-imaging agents. A neutral amyloid probe, (E,E)-1-(3′,4′-dihydroxystyryl)-4-(3′-methoxy-4 ′-hydroxystyryl)benzene (3), devoid of any carboxylate groups has been designed and synthesized via a 12-step reaction sequence with a total yield of 30%. The unsymmetric compound 3 has also been labelled with C-11 via [11C]methyl iodide ([11C]CH3I) methylation of a symmetric 4,4′-dimesyl protected precursor followed by deprotection. Preliminary evaluation indicated that compound 3 selectively stained plaques and neurofibrillary tangles in post-mortem AD brain, and exhibited good binding affinity (Ki = 38 ± 8 nM) for Aβ(1-40) fibrils in vitro. In vivo pharmacokinetic studies indicated that [11C]3 exhibited higher brain uptake than its carboxylic acid analogs and good clearance from normal control mouse brain. [11C]3 also exhibited specific in vivo binding to pancreatic amyloid deposits in the NOR-beta transgenic mouse model. These results justify further investigation of 3 and similar derivatives as surrogate markers for in vivo quantitation of amyloid deposits. Copyright

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