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5-Hydroxymethyldeoxyuridylate (5-HMdU) is a modified nucleoside found in DNA, resulting from the oxidation of 5-methylcytosine (5-mC). This chemical modification plays a significant role in epigenetic regulation, as it can influence gene expression without altering the DNA sequence. 5-HMdU is formed through the action of the enzyme TET (ten-eleven translocation), which converts 5-mC to 5-hydroxymethylcytosine (5-hmC) and further oxidizes it to 5-formylcytosine (5-fC) and eventually to 5-carboxylcytosine (5-caC). The presence of 5-HMdU in DNA can affect the binding of proteins and the structure of DNA, thereby impacting gene expression and cellular function. It has been implicated in various biological processes, including DNA repair, transcriptional regulation, and the maintenance of genomic stability.

5238-86-8

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5238-86-8 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 5238-86-8 includes 7 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 4 digits, 5,2,3 and 8 respectively; the second part has 2 digits, 8 and 6 respectively.
Calculate Digit Verification of CAS Registry Number 5238-86:
(6*5)+(5*2)+(4*3)+(3*8)+(2*8)+(1*6)=98
98 % 10 = 8
So 5238-86-8 is a valid CAS Registry Number.

5238-86-8SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 17, 2017

Revision Date: Aug 17, 2017

1.Identification

1.1 GHS Product identifier

Product name 5-hydroxymethyl-dUMP

1.2 Other means of identification

Product number -
Other names 5-hydroxymethyl-2'-deoxyuridine-5'-monophosphate

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:5238-86-8 SDS

5238-86-8Relevant academic research and scientific papers

Thymidine radical formation via one-electron transfer oxidation photoinduced by pterin: Mechanism and products characterization

Serrano, Mariana P.,Vignoni, Mariana,Lorente, Carolina,Vicendo, Patricia,Oliveros, Esther,Thomas, Andrés H.

, p. 418 - 431 (2016/06/01)

UV-A radiation (320-400 nm), recognized as a class I carcinogen, induces damage to the DNA molecule and its components through different mechanisms. Pterin derivatives are involved in various biological functions, including enzymatic processes, and it has been demonstrated that oxidized pterins may act as photosensitizers. In particular, they accumulate in the skin of patients suffering from vitiligo, a chronic depigmentation disorder. We have investigated the ability of pterin (Ptr), the parent compound of oxidized pterins, to photosensitize the degradation of the pyrimidine nucleotide thymidine 5′-monophosphate (dTMP) in aqueous solutions under UV-A irradiation. Although thymine is less reactive than purine nucleobases, our results showed that Ptr is able to photoinduce the degradation of dTMP and that the process is initiated by an electron transfer from the nucleotide to the triplet excited state of Ptr. In the presence of molecular oxygen, the photochemical process leads to the oxidation of dTMP, whereas Ptr is not consumed. In the absence of oxygen, both compounds are consumed to yield a product in which the pterin moiety is covalently linked to the thymine. This compound retains some of the spectroscopic properties of Ptr, such as absorbance in the UV-A region and fluorescence properties.

Trapping of an intermediate in the reaction catalyzed by flavin-dependent thymidylate synthase

Mishanina, Tatiana V.,Koehn, Eric M.,Conrad, John A.,Palfey, Bruce A.,Lesley, Scott A.,Kohen, Amnon

experimental part, p. 4442 - 4448 (2012/04/23)

Thymidylate is a DNA nucleotide that is essential to all organisms and is synthesized by the enzyme thymidylate synthase (TSase). Several human pathogens rely on an alternative flavin-dependent thymidylate synthase (FDTS), which differs from the human TSase both in structure and molecular mechanism. It has recently been shown that FDTS catalysis does not rely on an enzymatic nucleophile and that the proposed reaction intermediates are not covalently bound to the enzyme during catalysis, an important distinction from the human TSase. Here we report the chemical trapping, isolation, and identification of a derivative of such an intermediate in the FDTS-catalyzed reaction. The chemically modified reaction intermediate is consistent with currently proposed FDTS mechanisms that do not involve an enzymatic nucleophile, and it has never been observed during any other TSase reaction. These findings establish the timing of the methylene transfer during FDTS catalysis. The presented methodology provides an important experimental tool for further studies of FDTS, which may assist efforts directed toward the rational design of inhibitors as leads for future antibiotics.

Oxidation of nucleic acid related compounds by the peroxodisulfate ion

Itahara,Yoshitake,Koga,Nishino

, p. 2257 - 2264 (2007/10/02)

The treatment of nucleic acid bases, nucleosides, and nucleotides with peroxodisulfate ion in a phosphate buffer solution at pH 7.0 or water at 70-75°C was investigated. The reaction of thymine and 5-methylcytosine nucleosides and nucleotides resulted in the oxidation of the 5-methyl groups. The oxidation products from 1,3-dimethyluracils and the time-course of the reaction of uracils led to two plausible reaction mechanisms for the oxidation of uracils.

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