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(4-BENZOYL-PHENOXY)-ACETIC ACID, commonly known as fenbufen, is a nonsteroidal anti-inflammatory drug (NSAID) characterized by its phenylacetic acid derivative structure. It possesses analgesic, anti-inflammatory, and antipyretic properties, making it a valuable pharmaceutical agent for managing pain and inflammation associated with various conditions.

6322-83-4

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6322-83-4 Usage

Uses

Used in Pharmaceutical Industry:
(4-BENZOYL-PHENOXY)-ACETIC ACID is used as an anti-inflammatory agent for the treatment of inflammatory conditions such as rheumatoid arthritis, osteoarthritis, and other musculoskeletal disorders. It functions by inhibiting the production of inflammatory prostaglandins, which are hormones responsible for inducing pain and swelling in the body.
Used in Pain Management:
(4-BENZOYL-PHENOXY)-ACETIC ACID is utilized as an analgesic to alleviate pain associated with various conditions, including but not limited to musculoskeletal disorders.
Used in Antipyretic Therapy:
(4-BENZOYL-PHENOXY)-ACETIC ACID is employed as an antipyretic to reduce fever by counteracting the effects of prostaglandins that are involved in the regulation of body temperature.
Formulations:
(4-BENZOYL-PHENOXY)-ACETIC ACID is available in various forms, including tablets and topical preparations, offering flexibility in administration routes for patients.
Safety Considerations:
While generally well-tolerated, (4-BENZOYL-PHENOXY)-ACETIC ACID may cause gastrointestinal irritation, and its use should be approached with caution in individuals with a history of ulcers or gastrointestinal bleeding.

Check Digit Verification of cas no

The CAS Registry Mumber 6322-83-4 includes 7 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 4 digits, 6,3,2 and 2 respectively; the second part has 2 digits, 8 and 3 respectively.
Calculate Digit Verification of CAS Registry Number 6322-83:
(6*6)+(5*3)+(4*2)+(3*2)+(2*8)+(1*3)=84
84 % 10 = 4
So 6322-83-4 is a valid CAS Registry Number.
InChI:InChI=1/C15H12O4/c16-14(17)10-19-13-8-6-12(7-9-13)15(18)11-4-2-1-3-5-11/h1-9H,10H2,(H,16,17)

6322-83-4SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 14, 2017

Revision Date: Aug 14, 2017

1.Identification

1.1 GHS Product identifier

Product name 2-(4-benzoylphenoxy)acetic acid

1.2 Other means of identification

Product number -
Other names WLN: QV1OR DVR

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:6322-83-4 SDS

6322-83-4Relevant academic research and scientific papers

Fibrate-based N-acylsulphonamides targeting carbonic anhydrases: synthesis, biochemical evaluation, and docking studies

Ammazzalorso, Alessandra,Carradori, Simone,Angeli, Andrea,Akdemir, Atilla,De Filippis, Barbara,Fantacuzzi, Marialuigia,Giampietro, Letizia,Maccallini, Cristina,Amoroso, Rosa,Supuran, Claudiu T.

, p. 1051 - 1061 (2019/05/22)

A large library of fibrate-based N-acylsulphonamides was designed, synthesised, and fully characterised in order to propose them as zinc binders for the inhibition of human carbonic anhydrase (hCA) enzymatic activity. Synthesised compounds were tested aga

ACTIVATOR OF ADIPONECTIN RECEPTOR

-

Paragraph 0336; 0337, (2016/08/17)

An AdipoR activator for activating both AdipoR1 and AdipoR2 is provided. A compound represented by the following formula (1), wherein A is a substituted or unsubstituted aryl group or the like, Y1 is (CHR2)a— or the like, X is CH or N, R1 is a C1-7 alkyl group, m is an integer of 0-4, Y2 is *—O—CH2—CONH—, *—CONH—(CH2)b—CO— or the like, Z is a cyclic group, B may be a substituent of the cyclic group represented by Z, and n is an integer of 0-3.

Studies on the collision-induced dissociation of adipoR agonists after electrospray ionization and their implementation in sports drug testing

Dib, Josef,Schl?rer, Nils,Sch?nzer, Wilhelm,Thevis, Mario

, p. 407 - 417 (2015/03/30)

AdipoR agonists are small, orally active molecules capable of mimicking the protein adiponectin, which represents an adipokine with antidiabetic and antiatherogenic effects. Two adiponectin receptors were reported in the literature referred to as adipoR1 and adipoR2. Activation of these receptors stimulates mitochondrial biogenesis and results in an improved oxidative metabolism (via adipoR1) and increased insulin sensitivity (via adipoR2). Hence, adipoR agonists are potentially performance enhancing substances and targets of proactive and preventive anti-doping measures. In this study, two adipoR agonists termed AdipoRon and 112254 as well as two isotopically labeled internal standards (ISTDs) were synthesized in three-step reactions. The products were fully characterized by nuclear magnetic resonance spectroscopy (NMR), mass spectrometry (MS) and density functional theory (DFT) computation. Collision-induced dissociation pathways following electrospray ionization were suggested based on the determined elemental compositions of product ions, comparison to product ions derived from labeled analogs (ISTDs), H/D-exchange experiments and the results of DFT calculations. The most abundant product ions were found at m/z 174, tentatively assigned to protonated 1-benzyl-1,2,3,4-tetrahydropyridine for AdipoRon, and m/z 207, suggested as protonated 1-(4-methoxybenzyl)piperazine, for 112254. Notably, the loss of the heterocyclic ring (i.e. piperazine and piperidine, respectively) in a supposedly intramolecular elimination reaction was observed in both cases. A qualitative determination of both AdipoR agonists in human plasma was established and fully validated for doping control purposes. Validation items such as recovery (86-89%), specificity, linearity, lower limit of detection (1ng/ml), intraday (3-18%) and interday (5-16%) precision as well as ion suppression or enhancement were determined. Based on these findings adipoR agonists can be implemented in sports drug testing procedures.

Synthesis and biological evaluation of negative allosteric modulators of the Kv11.1(hERG) channel

Yu, Zhiyi,Van Veldhoven, Jacobus P.D.,'T Hart, Ingrid M.E.,Kopf, Adrian H.,Heitman, Laura H.,Ijzerman, Adriaan P.

, p. 50 - 59 (2015/11/23)

We synthesized and evaluated a series of compounds for their allosteric modulation at the Kv11.1 (hERG) channel. Most compounds were negative allosteric modulators of [3H]dofetilide binding to the channel, in particular 7f, 7h-j and 7p. Compounds 7f and 7p were the most potent negative allosteric modulators amongst all ligands, significantly increasing the dissociation rate of dofetilide in the radioligand kinetic binding assay, while remarkably reducing the affinities of dofetilide and astemizole in a competitive displacement assay. Additionally, both 7f and 7p displayed peculiar displacement characteristics with Hill coefficients significantly distinct from unity as shown by e.g., dofetilide, further indicative of their allosteric effects on dofetilide binding. Our findings in this investigation yielded several promising negative allosteric modulators for future functional and clinical research with respect to their antiarrhythmic propensities, either alone or in combination with known Kv11.1 blockers.

Effect of stilbene and chalcone scaffolds incorporation in clofibric acid on PPARα agonistic activity

Giampietro, Letizia,D'Angelo, Alessandra,Giancristofaro, Antonella,Ammazzalorso, Alessandra,De Filippis, Barbara,Di Matteo, Mauro,Fantacuzzi, Marialuigia,Linciano, Pasquale,Maccallini, Cristina,Amoroso, Rosa

, p. 59 - 65 (2014/01/17)

In an effort to develop safe and efficacious compounds for the treatment of metabolic disorders, new compounds based on a combination of clofibric acid, the active metabolite of clofibrate, and trans-stilbene, chalcone, and other lipophilic groups were synthesized. They were evaluated for PPARα transactivation activity; all branched derivatives showed an increase of the transcriptional activity of receptor compared to the linear ones. Noteworthy, stilbene and benzophenone branched derivatives activated the PPARα better than clofibric acid.

Benzhydrylamine linker grafting: A strategy for the improved synthesis of C-terminal peptide amides

Alewood, Dianne,Hopping, Gene,Brust, Andreas,Reid, Robert C.,Alewood, Paul F.

experimental part, p. 551 - 557 (2011/11/12)

The standard p-MBHA resin used during Boc-chemistry synthesis of peptides carrying C-terminal carboxamides is compromised by batch-to-batch variations in its performance. This can cause artificially 'difficult' couplings during peptide chain assembly, which may ultimately lead to failed syntheses given the inability to achieve acceptable coupling yields. To overcome these problems, we have developed a new approach by grafting a functionalized benzhydrylamine linker onto well-characterized and well-performing PAM resins. We combine optimized Boc-chemistry, high-performing PAM resins and new benzhydrylamine-based linkers to achieve improved syntheses of peptide amides. Here we present the synthesis of two new benzhydrylamine linkers and their attachment to selected PAM resins. This novel solid support was evaluated through the synthesis of selected 'difficult' conotoxins and monitoring the coupling efficiency using quantitative ninhydrin assay. The results show a superior performance of the novel linker solid support compared to the standard p-MBHA resins routinely used. In summary, we describe an alternative linker-resin system that allows improved access to C-terminal amide peptides employing Boc/Bzl chemistry. Benzhydrylamine enzhydrylamine linkers (a/b) were grafted onto high-performing Phe-PAM resins for Boc solid phase peptide amide synthesis. Fast couplings with high fidelity were achieved enabling the synthesis of selected conotoxins that previously demonstrated artificially 'difficult sequences' on commercially available p-MBHA resins. Copyright

Synthesis of a thioester linker precursor for a general preparation of peptide C-terminal thioacids

Gaertner, Hubert,Villain, Matteo,Botti, Paolo,Canne, Lynne

, p. 2239 - 2241 (2007/10/03)

A general procedure to prepare peptide thioacids by solid-phase peptide synthesis is presented. The method involves the synthesis of 4-[α-(S-acetyl)mercaptobenzyl]phenoxyacetic acid as general precursor. This reagent once attached to a solid support is de

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