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ethyl 2-[4-(4-fluorophenyl)piperazin-1-yl]acetate is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

68104-71-2

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68104-71-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 68104-71-2 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 6,8,1,0 and 4 respectively; the second part has 2 digits, 7 and 1 respectively.
Calculate Digit Verification of CAS Registry Number 68104-71:
(7*6)+(6*8)+(5*1)+(4*0)+(3*4)+(2*7)+(1*1)=122
122 % 10 = 2
So 68104-71-2 is a valid CAS Registry Number.

68104-71-2Relevant academic research and scientific papers

Design and synthesis of piperazine acetate podophyllotoxin ester derivatives targeting tubulin depolymerization as new anticancer agents

Sun, Wen-Xue,Ji, Ya-Jing,Wan, Yun,Han, Hong-Wei,Lin, Hong-Yan,Lu, Gui-Hua,Qi, Jin-Liang,Wang, Xiao-Ming,Yang, Yong-Hua

, p. 4066 - 4074 (2017)

In this paper, a series of podophyllotoxin piperazine acetate ester derivatives were synthesized and investigated due to their antiproliferation activity on different human cancer cell lines. Among the congeners, C5 manifested prominent cytotoxicity towar

Microwave-Assisted Synthesis, Antioxidant, and Antimicrobial Evaluation of Piperazine-Azole-Fluoroquinolone Based 1,2,4-Triazole Derivatives

Basoglu Ozdemir, Serap,Demirbas, Neslihan,Demirbas, Ahmet,Ayaz, Faik Ahmet,?olak, Nesrin

, p. 2744 - 2759 (2018/10/25)

Azole derivatives (10a–f) obtained starting from 1-(4-fluorohenyl)piperazine were converted to the corresponding Mannich bases (7a–d, 12a,b, and 16a,b) containing β-lactame or flouroquinolone core via a one-pot three-component reaction. The synthesis of c

INHIBITORS OF DIHYDROCERAMIDE DESATURASE FOR TREATING DISEASE

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Paragraph 00285, (2018/09/11)

Disclosed herein are dihydroceramide desaturase 1 (Des1) inhibitor compounds and compositions, which are useful in the treatment of diseases, such as metabolic, cardiovascular, fibrotic, autoimmune/chronic inflammatory diseases, cystic fibrosis, various cancers, neurodegenerative diseases, lipid storage disorders, and ischemia/reperfusion injury, where inhibition of Des1 is expected to be therapeutic to a patient. Methods of inhibition of Des1 activity in a human or animal subject are also provided.

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