862011-06-1

Usage

Uses

Used in Pesticide Production:
Phenyl (5-cyanopyridin-2-yl)carbamate is used as an active ingredient in the production of pesticides, due to its ability to inhibit acetylcholinesterase, disrupting the nervous system of pests and leading to their death.
Used in Herbicide Production:
In the agricultural industry, phenyl (5-cyanopyridin-2-yl)carbamate is used as an active ingredient in herbicides to control the growth of unwanted plants by targeting their nervous system.
Used in Insecticide Production:
Phenyl (5-cyanopyridin-2-yl)carbamate is utilized as an active ingredient in insecticides to eliminate insects by inhibiting their acetylcholinesterase, causing paralysis and death.
Note: Due to its high toxicity and classification as a hazardous chemical, phenyl (5-cyanopyridin-2-yl)carbamate requires proper handling and disposal to prevent harm to human health and the environment.

Upstream product

• 4214-73-7 6-aminonicotinonitrile 
• 1885-14-9 phenyl chloroformate 

Relevant academic research and scientific papers

2-Formylpyridyl Ureas as Highly Selective Reversible-Covalent Inhibitors of Fibroblast Growth Factor Receptor 4

As part of a project to identify FGFR4 selective inhibitors, scaffold morphing of a 2-formylquinoline amide hit identified series of 2-formylpyridine ureas (2-FPUs) with improved potency and physicochemical properties. In particular, tetrahydronaphthyridi

FORMYLATED N-HETEROCYCLIC DERIVATIVES AS FGFR4 INHIBITORS

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Ring-fused bicyclic pyridyl derivatives as FGFR4 inhibitors

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SPIROCYCLIC PIPERIDINE DERIVATIVES AS TRPM 8 MODULATORS

The present invention provides Transient Receptor Potential subfamily M, member 8 (TRPM8) modulators of formula (I). In particular, compounds described herein are useful for treating or preventing diseases, conditions and/or disorders modulated by TRPM8.

Chk-1 inhibitors

This invention provides compounds and methods for treating cancer. More particularly, the invention provides compounds that inhibit Chk-1. These compounds potentiate the action of DNA-damaging agents such as chemotherapy and radiation therapy.