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2-(2-bromobenzyl)benzo[d]isothiazol-3(2H)-one 1,1-dioxide is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

876677-48-4

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876677-48-4 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 876677-48-4 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 8,7,6,6,7 and 7 respectively; the second part has 2 digits, 4 and 8 respectively.
Calculate Digit Verification of CAS Registry Number 876677-48:
(8*8)+(7*7)+(6*6)+(5*6)+(4*7)+(3*7)+(2*4)+(1*8)=244
244 % 10 = 4
So 876677-48-4 is a valid CAS Registry Number.

876677-48-4Downstream Products

876677-48-4Relevant academic research and scientific papers

Open saccharin-based secondary sulfonamides as potent and selective inhibitors of cancer-related carbonic anhydrase IX and XII isoforms

D'ascenzio, Melissa,Guglielmi, Paolo,Secci, Daniela,Carradori, Simone,Florio, Rosalba,Mollica, Adriano,Ceruso, Mariangela,Supuran, Claudiu T.,Akdemir, Atilla,Sobolev, Anatoly P.

, p. 51 - 59 (2016)

A large number of novel secondary sulfonamides based on the open saccharin scaffold were synthesized and evaluated as selective inhibitors of four different isoforms of human carbonic anhydrase (hCA I, II, IX and XII, EC 4.2.1.1). They were obtained by re

Design, synthesis and evaluation of N-substituted saccharin derivatives as selective inhibitors of tumor-associated carbonic anhydrase XII

D'Ascenzio, Melissa,Carradori, Simone,De Monte, Celeste,Secci, Daniela,Ceruso, Mariangela,Supuran, Claudiu T.

, p. 1821 - 1831 (2014/03/21)

A series of N-alkylated saccharin derivatives were synthesized and tested for the inhibition of four different isoforms of human carbonic anhydrase (CA, EC 4. 2.1.1): the transmembrane tumor-associated CA IX and XII, and the cytosolic CA I and II. Most of the reported derivatives inhibited CA XII in the nanomolar/low micromolar range, hCA IX with KIs ranging between 11 and 390 nM, whereas they were inactive against both CA I (KIs >50 μM) and II (KIs ranging between 39.1 nM and 50 μM). Since CA I and II are off-targets of antitumor carbonic anhydrase inhibitors (CAIs), the obtained results represent an encouraging achievement for the development of new anticancer candidates without the common side effects of non-selective CAIs. Moreover, the lack of an explicit zinc binding function on these inhibitors opens the way towards the exploration of novel mechanisms of inhibition that could explain the high selectivity of these compounds for the inhibition of the transmembrane, tumor-associated isoforms over the cytosolic ones.

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