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5-methyl-3-phenyl-4,5-dihydro-1H-pyrazole is an organic compound with the molecular formula C11H12N2. It is a derivative of pyrazole, a five-membered heterocyclic ring containing two nitrogen atoms. The structure of 5-methyl-3-phenyl-4,5-dihydro-1H-pyrazole features a methyl group (-CH3) at the 5-position, a phenyl group (C6H5) at the 3-position, and a hydrogen atom at the 1-position. The compound is classified as a dihydro-pyrazole, indicating the presence of a double bond between two carbon atoms in the ring, which has been reduced to a single bond, resulting in the addition of two hydrogen atoms. This chemical is primarily used in the synthesis of various pharmaceuticals and agrochemicals due to its unique structure and reactivity.

939-04-8

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939-04-8 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 939-04-8 includes 6 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 3 digits, 9,3 and 9 respectively; the second part has 2 digits, 0 and 4 respectively.
Calculate Digit Verification of CAS Registry Number 939-04:
(5*9)+(4*3)+(3*9)+(2*0)+(1*4)=88
88 % 10 = 8
So 939-04-8 is a valid CAS Registry Number.

939-04-8Relevant academic research and scientific papers

Preparation, Antiepileptic Activity, and Cardiovascular Safety of Dihydropyrazoles as Brain-Penetrant T-Type Calcium Channel Blockers

Remen, Lubos,Bezen?on, Olivier,Simons, Lloyd,Gaston, Rick,Downing, Dennis,Gatfield, John,Roch, Catherine,Kessler, Melanie,Mosbacher, Johannes,Pfeifer, Thomas,Grisostomi, Corinna,Rey, Markus,Ertel, Eric A.,Moon, Richard

, p. 8398 - 8411 (2016)

A series of dihydropyrazole derivatives was developed as potent, selective, and brain-penetrating T-type calcium channel blockers. An optimized derivative, compound 6c, was advanced to in vivo studies, where it demonstrated efficacy in the WAG/Rij rat model of generalized nonconvulsive, absence-like epilepsy. Compound 6c was not efficacious in the basolateral amygdala kindling rat model of temporal lobe epilepsy, and it led to prolongation of the PR interval in ECG recordings in rodents.

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