L-Triiodothyronine Liothyronine Sodium T3 / Cytomel CAS 55-06-1 CAS NO.55-06-1

quick detail:

liothyronine sodium

alias: cytomel t3 ; 3,3',5－triiodothyronine sodium

cas no: 55-06-1

molecular formula: c15h11i3nnao4

mw: 672.96

nolecular weight: 672.96
einecs: 200-223-5

appearence: white to beige powder

usage: liothyronine sodium is a synthetic variant of the human body's own thyroid hormone.

description:

t3 in the body is responsible for regulating the uptake of various nutrients into cells and into the mitochondria of those cells in order to effectively become utilized for the production and consumption of energy.

the mitochondria of every single cell in the body utilizes carbohydrates (primarily), fat, and even protein for the production of an energy source known as atp (adenosine triphosphate). through the intake of more t3, this production of atp will increase, leading to an increased rate of energy consumption in the form of fats, carbohydrates, and protein. hence, this is why the consumption of too much t3 without the use of anabolic steroids can result in muscle loss.

the bodybuilding and athletic world is attracted to the use of t3 as a physique and/or performance enhancing drug because of its capability to distinctly boost the body’s metabolism in the effort to metabolize body fat at a greater rate.

t3 is traditionally utilize during cutting, dieting, and/or pre-contest phases of training due to the universal goal of these phases to break down body fat, though in recent years cytomel has gained some popularity as a useful agent during bulking and mass gaining phases of training (normally in conjunction with anabolic steroids) in order to better efficiently process nutrients and/or to keep body fat levels down during periods of higher caloric intake.

cytomel (t3) is commonly used with anabolic steroids due to its significant impact on the body’s metabolism as a whole. it is very important to understand that t3 is indiscriminate in its metabolism boosting properties – it will increase the metabolism of fats, carbohydrates, and protein all equally.

therefore, beyond a particular dose of t3, there is an increased risk of muscle loss through increased turnover of protein. through the use of anabolic steroids and a properly adjusted diet, this muscle loss as a result of t3 can be prevented as a result of the nitrogen-retaining and protein sparing properties of theanabolic steroids.

cytomel (t3) is also commonly combined with other fat loss agents in order to increase its overall effect, as it does work synergistically with other fat loss agents.

applications:

high purity liothyronine sodium/cytomel t3/3,3',5－triiodothyronine sodium/cas no: 55-06-1 200-223-5 weight loss powders

liothyronine is the most potent form of thyroid hormone. chemically, it is nearly identical to triiodothyronine (t₃). as such, it acts on the body to increase the basal metabolic rate, affect protein synthesis and increase the body's sensitivity to catecholamines (such as adrenaline) by permissiveness. the thyroid hormones are essential to proper development and differentiation of all cells of the human body. these hormones also regulate protein, fat, and carbohydrate metabolism, affecting how human cells use energetic compounds.

in comparison to levothyroxine (t₄), liothyronine has a faster onset of action as well as a shorter biological half-life, which may be due to less plasma protein binding to thyroxine-binding globulin and transthyretin.

physicians can use this instead of or in addition to levothyroxine (t₄) for patients undergoing thyroid withdrawal. when a patient has thyroid cancer or graves' disease, ablation therapy with radioactive iodine can be used to remove any trace thyroid tissue. for ¹³¹i therapy to be effective, the trace thyroid tissue must be avid to iodine. the best method is to starve the tissue of iodine but this can lead to hypothyroid symptoms for the patient. withdrawal from levothyroxine can be done but it takes six weeks of withdrawal for the remaining thyroid tissue to be completely starved. six weeks is needed owing to levothyroxine's long half life. six weeks can be inconvenient for the patient and delay treatment. liothyronine instead can be taken and withdrawn from for two weeks to starve the thyroid tissue. this is much safer and more convenient than levothyroxine.