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Detail of "57469-78-0"

  • CAS Number:
  • 57469-78-0
  • Name:

  • Lysyl-ketoprofen

  • Superlist Name:
  • Ketoprofen lysinate
  • Molecular Structure:
  • Formula:
  • C16H14O3.C6H14N2O2
  • Molecular Weight:
  • 400.47
  • Synonyms:
  • L-Lysine, mono(3-benzoyl-alpha-methylbenzeneacetate);2-(3-benzoylphenyl)propanoic acid; (2R)-2,6-diaminohexanoic acid;Ketoprofen lysine;Ketoprofen lysine salt;Ketoprofen lysinate;
  • Boiling Point:
  • 431.3 °C at 760 mmHg
  • Flash Point:
  • 228.8 °C
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CAS No.57469-78-0 Ketoprofen lysinate

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CAS No.57469-78-0 Ketoprofen lysinate

Ketoprofen Lysinate Salt

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CAS No.57469-78-0 Ketoprofen lysinate

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CAS No.57469-78-0 Ketoprofen lysinate

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CAS No.57469-78-0 Ketoprofen lysinate

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CAS No.57469-78-0 Ketoprofen lysinate

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CAS No.57469-78-0 Ketoprofen lysinate

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CAS No.57469-78-0 Ketoprofen lysinate

Ketoprofen Lysine

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CAS No.57469-78-0 Ketoprofen lysinate

Assay:99%  Appearance:white or off...  Package:25kg/drum  Transportation:By sea /Air ...

Enterprise standard ,the content is more than 99%

Supplier:Wuhan Fortuna Chemical Co.,Ltd [ China (Mainland)]

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CAS No.57469-78-0 Ketoprofen lysinate

ketoprofen lysine

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CAS No.57469-78-0 Ketoprofen lysinate

Supplier:Technodrugs & Intermediates (P) Ltd [ India]

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Reference

Ketoprofen lysine salt inhibits disuse-induced osteopenia in a new non-traumatic immobilization model in the rat
Ketoprofen lysine salt inhibits disuse-induced osteopenia in a new non-traumatic immobilization model in the rat. Fiorentino, S.; Melillo, G.; Fedele, G.; Clavenna, G.; D'Agostino, C.; Mainetti, E.; Caselli, G. F. (Consorzio BIOLAQ, L'Aquila 67100, Italy). Pharmacological Research, 33(4/5), 277-281 (English) 1996 Academic. CODEN: PHMREP. ISSN: 1043-6618. DOCUMENT TYPE: Journal CA Section: 1 (Pharmacology) In this study, the role of a classical aryl propionic acid, ketoprofen lysine salt (KLS), was evaluated in a new model of disuse-induced osteoporosis in the rat. Tail immobilization evoked a time-dependent bone loss in the caudal vertebral bodies, measured densitometrically as a redn. of bone mineral d. (BMD) and content (BMC). KLS was administered s.c. once daily for 10 days at 0.5 mg/kg, a dose lower than that effective to elicit an anti-inflammatory response. Under these conditions, KLS completely abolished the BMD and BMC decrease obsd. in the caudal vertebrae after a 10-day immobilization, without affecting bone mass in normal (nonimmobilized) rats. 57469-78-0 is just another one chemical used in this study. These results suggest that KLS can exert, besides to its anti-inflammatory effect, an antiresorptive activity on bone that could be useful in the prevention of disuse-induced osteopenia. .
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