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Detail of "8059-24-3"

  • CAS Number:
  • 8059-24-3
  • Name:

  • Vitamin B6

  • Deleted CAS:
  • 12001-78-4
  • Synonyms:
  • Adermine;Vitamin B6(Pyridoxine Hydrochloride);
  • EINECS:
  • 232-503-8
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CAS No.8059-24-3 Vitamin B6Competitive Product

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CAS No.8059-24-3 Vitamin B6Competitive Product

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CAS No.8059-24-3 Vitamin B6

PYRIDOXINE---We supply this product in very competitive price.

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CAS No.8059-24-3 Vitamin B6

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CAS No.8059-24-3 Vitamin B6

  Appearance:colorless to...  Package:25kgs/drum

product: Pyridoxine(VB6) Melting Point: 232-233℃ Water Solubility : 0.2 mg/mL Hazard Class: Non-dangerous goods Quantity/20'FCL: N/A Application: Pyridoxine can be used as processing agent in food industry according to GB2760-90. It can prevent skin disease, promote lipid m

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CAS No.8059-24-3 Vitamin B6

Vitanmin B6 HCL

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CAS No.8059-24-3 Vitamin B6

Test item Test Standard Test results Character crystalline powder Consistent Content 98.0%~100.5% 99.5% Residue burning ≤0.20% 0.1% Loss on drying ≤ 2.0% 0.7% Chloride 16.9%~17.6% 17.2% Heavy metal ≤ 0.1% 0.08% Conclusion The detection of the standard sample in line with

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CAS No.8059-24-3 Vitamin B6

Vitamin B6(Pyridoxine Hydrochloride) CAS NO. :8059-24-3 Molecular Formula :C8H11NO3.HCl Molecular Weight :205.64 (1)Take about 10mg of this product and add 100 ml of water to solve it. (2)Add this product into hydrochloric acid solution (9-1000) to make 10μg /ml s

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CAS No.8059-24-3 Vitamin B6

VITAMIN H

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Reference

The effect of vitamin B6 deficiency on alanine:glyoxylate aminotransferase isozymes in rat liver
The effect of vitamin B6 deficiency on alanine:glyoxylate aminotransferase isozymes in rat liver. Takada, Yoshikazu; Mori, Toshio; Noguchi, Tomoo (Dep. Biochem., Kyushu Dent. Coll., Kitakyushu 803, Japan). Arch. Biochem. Biophys., 229(1), 1-6 (English) 1984. CODEN: ABBIA4. ISSN: 0003-9861. DOCUMENT TYPE: Journal CA Section: 18 (Animal Nutrition) Alanine:glyoxylate aminotransferase (AGT) [9015-67-2] isoenzyme AGT 1 was located in rat liver peroxisomes and mitochondria, and AGT 2 only in the mitochondria. The holo activity of combined peroxisomal and mitochondrial AGT 1, with a low Michaelis const. (Km) for L-alanine, rapidly decreased after a lag time of ~2 days during feeding of a vitamin B6 [8059-24-3]-deficient diet (by 50% in 5 days and by 86% in 14 days). Peroxisomal AGT 1 activity was more affected than the mitochondrial. The holo activity of AGT 2, with a high Km for L-alanine, decreased more slowly than AGT 1 (by 33% in 14 days and by 60% in 28 days). Urinary excretion of oxalate began to increase in 8-9 days when AGT 2 remained intact, but most of the AGT 1 was depleted. When the defect in glyoxylate transamination in vivo in vitamin B6 deficiency is considered, these findings suggest that it is due to the deficiency of AGT 1. 9015-67-2 and 8059-24-3 are just another two chemicals used in this study. The importance of peroxisomal AGT 1 is discussed, since peroxisomes have been described to be probably the major site of glyoxylate formation. .
Factors affecting the accumulation of homocyst(e)ine in rats deficient in vitamin B-6
Factors affecting the accumulation of homocyst(e)ine in rats deficient in vitamin B-6. Smolin, Lori A.; Benevenga, Norlin J. (Dep. Nutr. Sci., Univ. Wisconsin, Madison, WI 53706, USA). J. Nutr., 114(1), 103-11 (English) 1984. CODEN: JONUAI. ISSN: 0022-3166. DOCUMENT TYPE: Journal CA Section: 18 (Animal Nutrition) Factors that may affect the concn. of accumulated homocysteine [6027-13-0] in rats deficient in vitamin B6 [8059-24-3] were studied. Hepatic cystathionine synthase activity in vitamin B6-deficient rats was 41.5% of control and plasma protein-bound and free homocysteine concns. were significantly increased; however, among deficient rats there was no correlation between the concn. of homocysteine and the activity of hepatic cystathionine synthase. 8059-24-3 and 6027-13-0 which are cas registry numbers are also used here. The plasma protein-bound homocysteine concn. did not fluctuate with time of day, but a decrease in bound homocysteine was found when blood was sampled repeatedly from the same animal during a 24-h period. A 24-h fast resulted in a redn. in plasma homocysteine to concns. not different from those of control animals. The protein content of the diet affected growth but not food intake of rats. Feeding a 10% vs. a 60% casein diet did not change the concn. of accumulated homocysteine. Of the factors examd., only food restriction significantly affected plasma homocysteine concn. in rats deficient in vitamin B6. It is speculated that decreased and sporadic food intake in rats deficient in vitamin B6 may be responsible for the large fluctuations in homocysteine concn. obsd. in these animals over time. .
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