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1024276-54-7

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1024276-54-7 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1024276-54-7 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,0,2,4,2,7 and 6 respectively; the second part has 2 digits, 5 and 4 respectively.
Calculate Digit Verification of CAS Registry Number 1024276-54:
(9*1)+(8*0)+(7*2)+(6*4)+(5*2)+(4*7)+(3*6)+(2*5)+(1*4)=117
117 % 10 = 7
So 1024276-54-7 is a valid CAS Registry Number.

1024276-54-7Relevant academic research and scientific papers

Design and synthesis of aryl sulfonamide-based nonsteroidal mineralocorticoid receptor antagonists

Futatsugi, Kentaro,Piotrowski, David W.,Casimiro-Garcia, Agustin,Robinson, Shaughn,Sammons, Matthew,Loria, Paula M.,Banker, Mary E.,Petersen, Donna N.,Schmidt, Natalia J.

, p. 6239 - 6242 (2013/11/19)

Hit-to-lead medicinal chemistry efforts are described starting from a screening hit 1, leading to a new class of aryl sulfonamide-based MR antagonist, exemplified by 17, that possesses favourable MR binding affinity, selectivity profile against closely related NHRs, physicochemical properties and metabolic stability.

Discovery and optimisation of a selective non-steroidal glucocorticoid receptor antagonist

Brown, Angus R.,Bosies, Michael,Cameron, Helen,Clark, John,Cowley, Angela,Craighead, Mark,Elmore, Moira A.,Firth, Alistair,Goodwin, Richard,Goutcher, Susan,Grant, Emma,Grassie, Morag,Grove, Simon J.A.,Hamilton, Niall M.,Hampson, Hannah,Hillier, Alison,Ho, Koc-Kan,Kiczun, Michael,Kingsbury, Celia,Kultgen, Steven G.,Littlewood, Peter T.A.,Lusher, Scott J.,MacDonald, Susan,McIntosh, Lorraine,McIntyre, Theresa,Mistry, Ashvin,Morphy, J. Richard,Nimz, Olaf,Ohlmeyer, Michael,Pick, Jack,Rankovic, Zoran,Sherborne, Brad,Smith, Alasdair,Speake, Michael,Spinks, Gayle,Thomson, Fiona,Watson, Lynn,Weston, Mark

scheme or table, p. 137 - 140 (2011/02/27)

High-throughput screening of 3.87 million compounds delivered a novel series of non-steroidal GR antagonists. Subsequent rounds of optimisation allowed progression from a non-selective ligand with a poor ADMET profile to an orally bioavailable, selective, stable, glucocorticoid receptor antagonist.

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