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trans-4-(2,2-dimethyl-1-oxopropoxy)-L-proline methyl ester 4-methylbenzenesulfonate salt is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

126111-10-2

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126111-10-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 126111-10-2 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 1,2,6,1,1 and 1 respectively; the second part has 2 digits, 1 and 0 respectively.
Calculate Digit Verification of CAS Registry Number 126111-10:
(8*1)+(7*2)+(6*6)+(5*1)+(4*1)+(3*1)+(2*1)+(1*0)=72
72 % 10 = 2
So 126111-10-2 is a valid CAS Registry Number.

126111-10-2Downstream Products

126111-10-2Relevant academic research and scientific papers

Angiotensin II antagonists

-

, (2008/06/13)

This invention provides novel heterocyclic derivatives, their pharmaceutical formulations, and their use for antagonizing angiotensin II receptors in mammals.

Angiotensin II antagonist intermediates

-

, (2008/06/13)

This invention provides novel heterocyclic derivatives, their pharmaceutical formulations, and their use for antagonizing angiotensin II receptors in mammals.

(Phosphinyloxy)acid amino acid inhibitors of angiotensin converting enzyme. 2. Terminal amino acid analogues of (S)-1-[6-Amino-2-[[hydroxy(4-phenylbutyl)phosphinyl]oxy]-1-oxohexyl]- L-proline

Karanewsky,Badia,Cushman,DeForrest,Dejneka,Lee,Loots,Petrillo

, p. 1459 - 1469 (2007/10/02)

Analogues of (S)-1-[6-amino-2-[[hydroxy(4-phenylbutyl)phosphinyl]oxy]-1-oxohexyl]- L-proline (1, SQ 29,852) in which the terminal proline residue has been replaced by a variety of substituted and heteroatom-substituted prolines, N-arylglycines, N-cycloalkylglycines, and bicyclic amino acids have been synthesized and evaluated as inhibitors of angiotensin converting enzyme in vitro an in vivo. In general, the addition of lipophilic substituents to the 4-position of proline of the parent phosphate 1 resulted in substantial increases in vitro activity. The largest improvements were observed in the case of cis-benzyl (36-fold) and dithioketal (24-fold) analogues 2r and 2x, respectively. These enhancements of in vitro activity were accompanied by modest increases (2-3.5-fold) in in vivo (iv) activity. Among the various terminal amino acid replacements examined in this study, the indoline-based analogue 2i was by far the most potent compound on iv administration in the normotensive rat.

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