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5-hydroxy-2-(4-chlorophenyl)benzofuran is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

1448962-34-2

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1448962-34-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 1448962-34-2 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,4,4,8,9,6 and 2 respectively; the second part has 2 digits, 3 and 4 respectively.
Calculate Digit Verification of CAS Registry Number 1448962-34:
(9*1)+(8*4)+(7*4)+(6*8)+(5*9)+(4*6)+(3*2)+(2*3)+(1*4)=202
202 % 10 = 2
So 1448962-34-2 is a valid CAS Registry Number.

1448962-34-2Relevant academic research and scientific papers

Synthesis of Benzofurans from Ketones and 1,4-Benzoquinones

Wu, Fengtian,Bai, Rongxian,Gu, Yanlong

, p. 2307 - 2316 (2016)

Benzofuran derivatives can be synthesized through the sequential Michael addition and cyclization of 1,3-dicarbonyl compounds with 1,4-benzoquinones. However, ketones are rarely used in this reaction because of their low nucleophilicities. In this study, this problem was solved by utilizing triethyl orthoformate, which enabled the formation of a vinyl ethyl ether as an additive. As a result, the nucleophilicity of ketones increased. Many important 5-hydroxybenzofuran derivatives, which were not readily available by synthesis in the past, were also prepared via these newly established reactions. (Figure presented.) .

Benzofuran derivatives as anticancer inhibitors of mTOR signaling

Salomé, Christophe,Ribeiro, Nigel,Chavagnan, Thierry,Thuaud, Frédéric,Serova, Maria,De Gramont, Armand,Faivre, Sandrine,Raymond, Eric,Désaubry, Laurent

, p. 181 - 191 (2014/06/09)

A series of 32 derivatives and isosteres of the mTOR inhibitor 2 were synthesized and compared for their cytotoxicity in radioresistant SQ20B cancer cell line. Several of these compounds, in particular 30b, were significantly more cytotoxic than 2. Importantly, 30b was shown to block both mTORC1 and Akt signaling, suggesting insensitivity to the resistance associated to Akt overactivation observed with rapamycin derivatives currently used in clinic.

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