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tetrachloro-cyclopentadienone-phenylhydrazone is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

17581-53-2

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17581-53-2 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 17581-53-2 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 1,7,5,8 and 1 respectively; the second part has 2 digits, 5 and 3 respectively.
Calculate Digit Verification of CAS Registry Number 17581-53:
(7*1)+(6*7)+(5*5)+(4*8)+(3*1)+(2*5)+(1*3)=122
122 % 10 = 2
So 17581-53-2 is a valid CAS Registry Number.

17581-53-2Downstream Products

17581-53-2Relevant academic research and scientific papers

INHIBITORS OF MTOR-DEPTOR INTERACTIONS AND METHODS OF USE THEREOF

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Page/Page column 35, (2018/05/24)

Provided herein are substituted hydrazone compounds useful as inhibitors of DEPTOR. The invention further provides pharmaceutical compositions of the compounds of the invention. The invention also provides medical uses of substituted hydrazone compounds.

Structure-activity relationship study of small molecule inhibitors of the DEPTOR-mTOR interaction

Lee, Jihye,Shi, Yijiang,Vega, Mario,Yang, Yonghui,Gera, Joseph,Jung, Michael E.,Lichtenstein, Alan

supporting information, p. 4714 - 4724 (2017/09/29)

DEPTOR is a 48 kDa protein that binds to mTOR and inhibits this kinase within mTORC1 and mTORC2 complexes. Over-expression of DEPTOR specifically occurs in the multiple myeloma (MM) tumor model and DEPTOR knockdown is cytotoxic to MM cells, suggesting it is a potential therapeutic target. Since mTORC1 paralysis protects MM cells against DEPTOR knockdown, it indicates that the protein–protein interaction between DEPTOR and mTOR is key to MM viability vs death. In a previous study, we used a yeast two-hybrid screen of a small inhibitor library to identify a compound that inhibited DEPTOR/mTOR binding in yeast. This therapeutic (compound B) also prevented DEPTOR/mTOR binding in MM cells and was selectively cytotoxic to MM cells. We now present a structure–activity relationship (SAR) study around this compound as a follow-up report of this previous work. This study has led to the discovery of five new leads – namely compounds 3g, 3k, 4d, 4e and 4g – all of which have anti-myeloma cytotoxic properties superior to compound B. Due to their targeting of DEPTOR, these compounds activate mTORC1 and selectively induce MM cell apoptosis and cell cycle arrest.

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