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N1,N2-bis(4-bromophenyl)oxalamide is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

21022-25-3

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21022-25-3 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 21022-25-3 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 2,1,0,2 and 2 respectively; the second part has 2 digits, 2 and 5 respectively.
Calculate Digit Verification of CAS Registry Number 21022-25:
(7*2)+(6*1)+(5*0)+(4*2)+(3*2)+(2*2)+(1*5)=43
43 % 10 = 3
So 21022-25-3 is a valid CAS Registry Number.

21022-25-3Relevant academic research and scientific papers

The effect of fluorine atom on the synthesis and composition of gametocidal ethyl oxanilates

Iskra, Jernej,Titan, Primoz,Meglic, Vladimir

, p. 561 - 568 (2013/09/23)

Three derivatives of ethyl oxanilate were synthesized in order to test their application as gametocides on the hermaphrodite plants like common wheat (Triticum aestivum L.). A substituent at para position (F, Br, CN) of aniline defined its reactivity towa

Synthesis of N,N′-diarylalkanediamides and their antimycobacterial and antialgal activity

Kubicova, Lenka,Waisser, Karel,Kunes, Jiri,Kralova, Katarina,Odlerova, Zelmira,Slosarek, Milan,Janota, Jiri,Svoboda, Zbynek

, p. 714 - 726 (2007/10/03)

A set of N,N′-diarylalkanediamides was synthesized. The compounds were tested for their antimycobacterial and antialgal activity. The antimycobacterial activity of N,N′-diarylalkanediamides depends on the lipophilicity of the respective acid. Antimycobacteri-ally active substances were found only in the series of N,N′-diarylethanediamides and N,N′-diarylbutanediamides. Other compounds (derivatives of pentane-, hexane-, octane- and nonanediamide) were inactive against various strains of mycobacteria. The compounds inhibited growth and chlorophyll production in Chlorella vulgaris. Their relatively low antial-gal activity is probably connected with their lowered aqueous solubility, and hence by a restricted passage of the inhibitor through the hydrophilic regions of thylakoid membranes.

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