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3,5-di-tert-butyl-4-hydroxybenzaldehyde dimethyl acetal is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

23093-16-5

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23093-16-5 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 23093-16-5 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 2,3,0,9 and 3 respectively; the second part has 2 digits, 1 and 6 respectively.
Calculate Digit Verification of CAS Registry Number 23093-16:
(7*2)+(6*3)+(5*0)+(4*9)+(3*3)+(2*1)+(1*6)=85
85 % 10 = 5
So 23093-16-5 is a valid CAS Registry Number.

23093-16-5SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 18, 2017

Revision Date: Aug 18, 2017

1.Identification

1.1 GHS Product identifier

Product name 3,5-di-tert-butyl-4-hydroxybenzaldehyde dimethyl acetal

1.2 Other means of identification

Product number -
Other names 3,5-Di-t-butyl-4-hydroxybenzaldehyd-dimethylacetal

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:23093-16-5 SDS

23093-16-5Relevant academic research and scientific papers

Discovery of a negative allosteric modulator of GABAB receptors

Chen, Lin-Hai,Sun, Bing,Zhang, Yang,Xu, Tong-Jie,Xia, Zhi-Xiong,Liu, Jian-Feng,Nan, Fa-Jun

, p. 742 - 747 (2014/08/05)

Initialized from the scaffold of CGP7930, an allosteric agonist of GABAB receptors, a series of noncompetitive antagonists were discovered. Among these compounds, compounds 3, 6, and 14 decreased agonist GABA-induced maximal effect of IP3 production in HEK293 cells overexpressing GABAB receptors and Gqi9 proteins without changing the EC50. Compounds 3, 6, and 14 not only inhibited agonist baclofen-induced ERK1/2 phosphorylation but also blocked CGP7930-induced ERK1/2 phosphorylation in HEK293 cells overexpressing GABAB receptors. The results suggested that compounds 3, 6, and 14 are negative allosteric modulators of GABAB receptors. The representative compound 14 decreased GABA-induced IP3 production with IC50 of 37.9 μM and had no effect on other GPCR Class C members such as mGluR1, mGluR2, and mGluR5. Finally, we showed that compound 14 did not bind to the orthosteric binding sites of GABAB receptors, demonstrating that compound 14 negatively modulated GABAB receptors activity as a negative allosteric modulator.

Highly E-selective and effective synthesis of antiarthritic drug candidate S-2474 using quinone methide derivatives

Inagaki, Masanao,Haga, Nobuhiro,Kobayashi, Makoto,Ohta, Naoki,Kamata, Susumu,Tsuri, Tatsuo

, p. 125 - 128 (2007/10/03)

We have developed an efficient and E-selective synthesis of an antiarthritic drug candidate (E)-(5)-(3,5-di-tert-butyl-4-hydroxybenzylidene)-2-ethyl- 1,2-isothiazolidine-1,1-dioxide (S-2474; 1), in which α-methoxy-p-quinone methide is used as a key intermediate. α-Methoxy-p-quinone methide was revealed to be an equivalent to a p-hydroxy protected benzaldehyde. It reacts smoothly with α-sulfonyl carbanion to give 1,6-addition intermediates, which can be further processed to provide S-2474 directly in the presence of a base. This procedure gives S-2474 as an almost single isomer on the benzylidene double bond in excellent yield and thus is a very practical method adaptable to large-scale synthesis. The detailed mechanistic aspects are studied and discussed.

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